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Gerald M Cohen

Showing results (1-10 of 50) with videos related to

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Biochimie|May 23, 2002
The Apaf-1 apoptosome: a large caspase-activating complexKelvin Cain, Shawn B Bratton, Gerald M Cohen
The Journal of Biological Chemistry|May 2, 2003
Fas-associated death domain protein and caspase-8 are not recruited to the tumor necrosis factor receptor 1 signaling complex during tumor necrosis factor-induced apoptosisNicholas Harper, Michelle Hughes, Marion MacFarlane, et al.
Cell Reports|October 15, 2013
BIM-mediated membrane insertion of the BAK pore domain is an essential requirement for apoptosisKathrin Weber, Nicholas Harper, John Schwabe, et al.
British Journal of Cancer|March 9, 2016
BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cellsMichael Butterworth, Andrew Pettitt, Shankar Varadarajan, et al.
Cell Death & Disease|July 10, 2019
Apogossypol-mediated reorganisation of the endoplasmic reticulum antagonises mitochondrial fission and apoptosisGovindaraju Yedida, Mateus Milani, Gerald M Cohen, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|October 2, 2007
Barriers to effective TRAIL-targeted therapy of malignancyMartin J S Dyer, Marion MacFarlane, Gerald M Cohen
The Journal of Biological Chemistry|December 15, 2005
Caspase-7 is directly activated by the approximately 700-kDa apoptosome complex and is released as a stable XIAP-caspase-7 approximately 200-kDa complexDavina Twiddy, Gerald M Cohen, Marion Macfarlane, et al.
Autophagy|December 2, 2010
The in vitro cleavage of the hAtg proteins by cell death proteasesJoanna M Norman, Gerald M Cohen, Edward T W Bampton
The Journal of Biological Chemistry|July 18, 2002
Proteasome-mediated degradation of Smac during apoptosis: XIAP promotes Smac ubiquitination in vitroMarion MacFarlane, Wendy Merrison, Shawn B Bratton, et al.
British Journal of Haematology|July 6, 2013
ABT-199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not plateletsMeike Vogler, David Dinsdale, Martin J S Dyer, et al.
Pageof 5

Showing results (1-10 of 50) with videos related to

Sort By:
Pageof 5
Biochimie|May 23, 2002
The Apaf-1 apoptosome: a large caspase-activating complexKelvin Cain, Shawn B Bratton, Gerald M Cohen
The Journal of Biological Chemistry|May 2, 2003
Fas-associated death domain protein and caspase-8 are not recruited to the tumor necrosis factor receptor 1 signaling complex during tumor necrosis factor-induced apoptosisNicholas Harper, Michelle Hughes, Marion MacFarlane, et al.
Cell Reports|October 15, 2013
BIM-mediated membrane insertion of the BAK pore domain is an essential requirement for apoptosisKathrin Weber, Nicholas Harper, John Schwabe, et al.
British Journal of Cancer|March 9, 2016
BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cellsMichael Butterworth, Andrew Pettitt, Shankar Varadarajan, et al.
Cell Death & Disease|July 10, 2019
Apogossypol-mediated reorganisation of the endoplasmic reticulum antagonises mitochondrial fission and apoptosisGovindaraju Yedida, Mateus Milani, Gerald M Cohen, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|October 2, 2007
Barriers to effective TRAIL-targeted therapy of malignancyMartin J S Dyer, Marion MacFarlane, Gerald M Cohen
The Journal of Biological Chemistry|December 15, 2005
Caspase-7 is directly activated by the approximately 700-kDa apoptosome complex and is released as a stable XIAP-caspase-7 approximately 200-kDa complexDavina Twiddy, Gerald M Cohen, Marion Macfarlane, et al.
Autophagy|December 2, 2010
The in vitro cleavage of the hAtg proteins by cell death proteasesJoanna M Norman, Gerald M Cohen, Edward T W Bampton
The Journal of Biological Chemistry|July 18, 2002
Proteasome-mediated degradation of Smac during apoptosis: XIAP promotes Smac ubiquitination in vitroMarion MacFarlane, Wendy Merrison, Shawn B Bratton, et al.
British Journal of Haematology|July 6, 2013
ABT-199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not plateletsMeike Vogler, David Dinsdale, Martin J S Dyer, et al.
Pageof 5