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Gerald M Cohen

Showing results (11-20 of 50) with videos related to

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Cancer Research|July 5, 2006
Inhibition of histone deacetylase class I but not class II is critical for the sensitization of leukemic cells to tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosisSatoshi Inoue, Antonello Mai, Martin J S Dyer, et al.
The American Journal of Pathology|January 27, 2004
Intermediate filaments control the intracellular distribution of caspases during apoptosisDavid Dinsdale, Justine C Lee, Grant Dewson, et al.
The Journal of Biological Chemistry|January 22, 2002
Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis proteinXiao-Ming Sun, Shawn B Bratton, Michael Butterworth, et al.
Molecular Cancer Therapeutics|November 6, 2009
Enhanced Fas-associated death domain recruitment by histone deacetylase inhibitors is critical for the sensitization of chronic lymphocytic leukemia cells to TRAIL-induced apoptosisSatoshi Inoue, Nick Harper, Renata Walewska, et al.
The Journal of Biological Chemistry|August 16, 2003
Protein kinase C modulates tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by targeting the apical events of death receptor signalingNicholas Harper, Michelle A Hughes, Stuart N Farrow, et al.
Neoplasia (New York, N.Y.)|May 2, 2013
Sabutoclax (BI97C1) and BI112D1, putative inhibitors of MCL-1, induce mitochondrial fragmentation either upstream of or independent of apoptosisShankar Varadarajan, Michael Butterworth, Jun Wei, et al.
The Journal of Biological Chemistry|March 15, 2006
p53 is cleaved by caspases generating fragments localizing to mitochondriaBerna S Sayan, A Emre Sayan, Richard A Knight, et al.
Molecular Cell|April 8, 2004
Caspase activation inhibits proteasome function during apoptosisXiao-Ming Sun, Michael Butterworth, Marion MacFarlane, et al.
The Journal of Biological Chemistry|March 1, 2007
Receptor-mediated endocytosis is not required for tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosisSusan L Kohlhaas, Andrew Craxton, Xiao-Ming Sun, et al.
Molecular Cell|August 18, 2009
Reconstitution of the death-inducing signaling complex reveals a substrate switch that determines CD95-mediated death or survivalMichelle A Hughes, Nicholas Harper, Michael Butterworth, et al.
Pageof 5

Showing results (11-20 of 50) with videos related to

Sort By:
Pageof 5
Cancer Research|July 5, 2006
Inhibition of histone deacetylase class I but not class II is critical for the sensitization of leukemic cells to tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosisSatoshi Inoue, Antonello Mai, Martin J S Dyer, et al.
The American Journal of Pathology|January 27, 2004
Intermediate filaments control the intracellular distribution of caspases during apoptosisDavid Dinsdale, Justine C Lee, Grant Dewson, et al.
The Journal of Biological Chemistry|January 22, 2002
Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis proteinXiao-Ming Sun, Shawn B Bratton, Michael Butterworth, et al.
Molecular Cancer Therapeutics|November 6, 2009
Enhanced Fas-associated death domain recruitment by histone deacetylase inhibitors is critical for the sensitization of chronic lymphocytic leukemia cells to TRAIL-induced apoptosisSatoshi Inoue, Nick Harper, Renata Walewska, et al.
The Journal of Biological Chemistry|August 16, 2003
Protein kinase C modulates tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by targeting the apical events of death receptor signalingNicholas Harper, Michelle A Hughes, Stuart N Farrow, et al.
Neoplasia (New York, N.Y.)|May 2, 2013
Sabutoclax (BI97C1) and BI112D1, putative inhibitors of MCL-1, induce mitochondrial fragmentation either upstream of or independent of apoptosisShankar Varadarajan, Michael Butterworth, Jun Wei, et al.
The Journal of Biological Chemistry|March 15, 2006
p53 is cleaved by caspases generating fragments localizing to mitochondriaBerna S Sayan, A Emre Sayan, Richard A Knight, et al.
Molecular Cell|April 8, 2004
Caspase activation inhibits proteasome function during apoptosisXiao-Ming Sun, Michael Butterworth, Marion MacFarlane, et al.
The Journal of Biological Chemistry|March 1, 2007
Receptor-mediated endocytosis is not required for tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced apoptosisSusan L Kohlhaas, Andrew Craxton, Xiao-Ming Sun, et al.
Molecular Cell|August 18, 2009
Reconstitution of the death-inducing signaling complex reveals a substrate switch that determines CD95-mediated death or survivalMichelle A Hughes, Nicholas Harper, Michael Butterworth, et al.
Pageof 5