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Neuroscience|July 2, 2016
The T-type calcium channel antagonist Z944 disrupts prepulse inhibition in both epileptic and non-epileptic ratsWendie N Marks, Quentin Greba, Stuart M Cain, et al.The Journal of Physiology|June 20, 2003
Functional roles of cytoplasmic loops and pore lining transmembrane helices in the voltage-dependent inactivation of HVA calcium channelsStephanie C Stotz, Scott E Jarvis, Gerald W ZamponiBiochemical and Biophysical Research Communications|July 19, 2011
Bipartite syntaxin 1A interactions mediate CaV2.2 calcium channel regulationJonathan N Davies, Scott E Jarvis, Gerald W ZamponiMolecular Brain|September 21, 2021
Splice-variant specific effects of a CACNA1H mutation associated with writer's crampIvana A Souza, Maria A Gandini, Gerald W ZamponiBehavioural Brain Research|March 29, 2011
Depressive-like behaviour of mice lacking cellular prion proteinVinicius M Gadotti, Stephan P Bonfield, Gerald W ZamponiMolecular Brain|April 30, 2014
Effect of the Brugada syndrome mutation A39V on calmodulin regulation of Cav1.2 channelsBrett A Simms, Ivana Assis Souza, Gerald W ZamponiPflugers Archiv : European Journal of Physiology|December 20, 2013
A novel calmodulin site in the Cav1.2 N-terminus regulates calcium-dependent inactivationBrett A Simms, Ivana Assis Souza, Gerald W ZamponiMolecular Brain|April 19, 2015
Glutamate receptors function as scaffolds for the regulation of β-amyloid and cellular prion protein signaling complexesAlison Hamilton, Gerald W Zamponi, Stephen S G FergusonChannels (Austin, Tex.)|August 10, 2013
Modeling interactions between voltage-gated Ca (2+) channels and KCa1.1 channelsJordan Dt Engbers, Gerald W Zamponi, Ray W TurnerBMC Genomics|September 27, 2025
Cataloging the potential functional diversity of Cacna1e splice variants using long-read sequencingShamsuddin A Bhuiyan, John R Tyson, Manuel Belmadani, et al.Pageof 44