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Kidney International|June 14, 2013
Increased lysosomal proteolysis counteracts protein accumulation in the proximal tubule during focal segmental glomerulosclerosisRikke Nielsen, Geraldine Mollet, Ernie L Esquivel, et al.Kidney International|October 17, 2017
Abolishment of proximal tubule albumin endocytosis does not affect plasma albumin during nephrotic syndrome in miceKathrin Weyer, Pia K Andersen, Kasper Schmidt, et al.Journal of the American Society of Nephrology : JASN|January 3, 2001
Structure of the human type IV collagen gene COL4A3 and mutations in autosomal Alport syndromeLaurence Heidet, Christelle Arrondel, Lionel Forestier, et al.Plos One|October 20, 2017
An inducible mouse model of podocin-mutation-related nephrotic syndromeMansoureh Tabatabaeifar, Tanja Wlodkowski, Ivana Simic, et al.JCI Insight|December 18, 2025
Splice modulation of COL4A5 reinstates collagen IV assembly in an organoid model of Alport syndromeHassan Saei, Bruno Estebe, Nicolas Goudin, et al.Journal of Cellular and Molecular Medicine|August 2, 2019
Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndromeAlexey Larionov, Eileen Dahlke, Madlen Kunke, et al.Nature Communications|October 28, 2022
A slit-diaphragm-associated protein network for dynamic control of renal filtrationMaciej K Kocylowski, Hande Aypek, Wolfgang Bildl, et al.Science Translational Medicine|August 9, 2023
Adeno-associated virus gene therapy prevents progression of kidney disease in genetic models of nephrotic syndromeWen Y Ding, Valeryia Kuzmuk, Sarah Hunter, et al.Plos Genetics|May 17, 2018
A homozygous KAT2B variant modulates the clinical phenotype of ADD3 deficiency in humans and fliesSara Gonçalves, Julie Patat, Maria Clara Guida, et al.Plos Genetics|October 27, 2018
Correction: A homozygous KAT2B variant modulates the clinical phenotype of ADD3 deficiency in humans and fliesSara Gonçalves, Julie Patat, Maria Clara Guida, et al.Pageof 2