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Plos Medicine|July 13, 2006
MPLW515L is a novel somatic activating mutation in myelofibrosis with myeloid metaplasiaYana Pikman, Benjamin H Lee, Thomas Mercher, et al.
Cell|March 3, 2015
Tuning cytokine receptor signaling by re-orienting dimer geometry with surrogate ligandsIgnacio Moraga, Gerlinde Wernig, Stephan Wilmes, et al.
Science Translational Medicine|November 30, 2018
Surgical adhesions in mice are derived from mesothelial cells and can be targeted by antibodies against mesothelial markersJonathan M Tsai, Rahul Sinha, Jun Seita, et al.
Science (New York, N.Y.)|April 23, 2021
Preventing Engrailed-1 activation in fibroblasts yields wound regeneration without scarringShamik Mascharak, Heather E desJardins-Park, Michael F Davitt, et al.
Blood|February 7, 2008
High-throughput sequence analysis of the tyrosine kinome in acute myeloid leukemiaMarc M Loriaux, Ross L Levine, Jeffrey W Tyner, et al.
Cancer Cell|April 9, 2008
Efficacy of TG101348, a selective JAK2 inhibitor, in treatment of a murine model of JAK2V617F-induced polycythemia veraGerlinde Wernig, Michael G Kharas, Rachel Okabe, et al.
Science Translational Medicine|March 12, 2025
Postoperative adhesions are abrogated by a sustained-release anti-JUN therapeutic in preclinical modelsDeshka S Foster, Jason L Guo, Emily Meany, et al.
Nature Communications|August 15, 2020
Elucidating the fundamental fibrotic processes driving abdominal adhesion formationDeshka S Foster, Clement D Marshall, Gunsagar S Gulati, et al.
Science Translational Medicine|September 13, 2021
JUN promotes hypertrophic skin scarring via CD36 in preclinical in vitro and in vivo modelsMichelle F Griffin, Mimi R Borrelli, Julia T Garcia, et al.
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