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Journal of Peptide Science : an Official Publication of the European Peptide Society|May 22, 2014
Aggregation propensity of Aib homo-peptides of different length: an insight from molecular dynamics simulationsGianfranco Bocchinfuso, Paolo Conflitti, Stefano Raniolo, et al.
Chemical Science|May 14, 2023
Isoamphipathic antibacterial molecules regulating activity and toxicity through positional isomerismSwagatam Barman, Sudip Mukherjee, Logia Jolly, et al.
Biochimica Et Biophysica Acta|April 29, 2009
Membrane perturbation by the antimicrobial peptide PMAP-23: a fluorescence and molecular dynamics studyBarbara Orioni, Gianfranco Bocchinfuso, Jin Young Kim, et al.
Human Mutation|May 9, 2018
Clinical and functional characterization of two novel ZBTB20 mutations causing Primrose syndromeEmilia Stellacci, Katharina Steindl, Pascal Joset, et al.
Advanced Healthcare Materials|September 26, 2017
Enhanced EGFR Targeting Activity of Plasmonic Nanostructures with Engineered GE11 PeptideFrancesca Biscaglia, Senthilkumar Rajendran, Paolo Conflitti, et al.
Clinical Genetics|December 23, 2020
Compound heterozygosity for PTPN11 variants in a subject with Noonan syndrome provides insights into the mechanism of SHP2-related disordersRebeca Lorca, Luca Pannone, Elías Cuesta-Llavona, et al.
Human Molecular Genetics|October 8, 2021
Caenorhabditis elegans provides an efficient drug screening platform for GNAO1-related disorders and highlights the potential role of caffeine in controlling dyskinesiaMartina Di Rocco, Serena Galosi, Enrico Lanza, et al.
Journal of Peptide Science : an Official Publication of the European Peptide Society|June 11, 2021
Formulation matters! A spectroscopic and molecular dynamics investigation on the peptide CIGB552 as itself and in its therapeutical formulationMarco Savioli, Lorenzo Antonelli, Gianfranco Bocchinfuso, et al.
American Journal of Human Genetics|June 15, 2006
Germline missense mutations affecting KRAS Isoform B are associated with a severe Noonan syndrome phenotypeClaudio Carta, Francesca Pantaleoni, Gianfranco Bocchinfuso, et al.
Human Molecular Genetics|March 29, 2008
Diverse driving forces underlie the invariant occurrence of the T42A, E139D, I282V and T468M SHP2 amino acid substitutions causing Noonan and LEOPARD syndromesSimone Martinelli, Paola Torreri, Michele Tinti, et al.
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