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Nucleus (Austin, Tex.)|February 18, 2011
The cellular response to DNA damage: a focus on MDC1 and its interacting proteinsGideon Coster, Michal GoldbergMethods in Cell Biology|February 15, 2024
Cloning and expansion of repetitive DNA sequencesSophie L Williams, Gideon CosterScience (New York, N.Y.)|July 22, 2017
Bidirectional eukaryotic DNA replication is established by quasi-symmetrical helicase loadingGideon Coster, John F X DiffleyDNA Repair|December 10, 2025
How DNA secondary structures drive replication fork instabilityAditya Sethi, María Fernández-Casañas, Billie Delpino, et al.The Journal of Biological Chemistry|September 4, 2012
A dual interaction between the DNA damage response protein MDC1 and the RAG1 subunit of the V(D)J recombinaseGideon Coster, Ayala Gold, Darlene Chen, et al.Nature Communications|July 19, 2022
The mechanism of replication stalling and recovery within repetitive DNACorella S Casas-Delucchi, Manuel Daza-Martin, Sophie L Williams, et al.Molecular Cell|August 5, 2014
Origin licensing requires ATP binding and hydrolysis by the MCM replicative helicaseGideon Coster, Jordi Frigola, Fabienne Beuron, et al.The Journal of Biological Chemistry|September 11, 2007
The DNA damage response mediator MDC1 directly interacts with the anaphase-promoting complex/cyclosomeGideon Coster, Zvi Hayouka, Liron Argaman, et al.Nucleic Acids Research|June 16, 2021
Cytosine base modifications regulate DNA duplex stability and metabolismCathia Rausch, Peng Zhang, Corella S Casas-Delucchi, et al.The EMBO Journal|October 2, 2023
Replication-induced DNA secondary structures drive fork uncoupling and breakageSophie L Williams, Corella S Casas-Delucchi, Federica Raguseo, et al.Pageof 1