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Traffic (Copenhagen, Denmark)|November 28, 2002
Inhibition of autophagy in mitotic animal cellsEeva-Liisa Eskelinen, Alan R Prescott, Jennifer Cooper, et al.Proceedings of the National Academy of Sciences of the United States of America|April 28, 2010
Phenothiazines inhibit S100A4 function by inducing protein oligomerizationVladimir N Malashkevich, Natalya G Dulyaninova, Udupi A Ramagopal, et al.The Journal of Cell Biology|September 1, 2004
Phospholipase C and cofilin are required for carcinoma cell directionality in response to EGF stimulationGhassan Mouneimne, Lilian Soon, Vera DesMarais, et al.Biochemistry|April 16, 2008
Structure of Ca2+-bound S100A4 and its interaction with peptides derived from nonmuscle myosin-IIAVladimir N Malashkevich, Kristen M Varney, Sarah C Garrett, et al.Science Signaling|August 18, 2016
Coincident signals from GPCRs and receptor tyrosine kinases are uniquely transduced by PI3Kβ in myeloid cellsDaniel M Houslay, Karen E Anderson, Tamara Chessa, et al.Proceedings of the National Academy of Sciences of the United States of America|April 1, 2015
Phosphatidylinositol 4-phosphate and phosphatidylinositol 3-phosphate regulate phagolysosome biogenesisAndreas Jeschke, Nicole Zehethofer, Buko Lindner, et al.Experimental Cell Research|March 5, 2008
RhoA/ROCK-mediated switching between Cdc42- and Rac1-dependent protrusion in MTLn3 carcinoma cellsMirvat El-Sibai, Olivier Pertz, Huan Pang, et al.Proceedings of the National Academy of Sciences of the United States of America|October 26, 2011
Myosin II isoform switching mediates invasiveness after TGF-β-induced epithelial-mesenchymal transitionJordan R Beach, George S Hussey, Tyler E Miller, et al.Proceedings of the National Academy of Sciences of the United States of America|December 1, 2011
Tripartite motif containing protein 27 negatively regulates CD4 T cells by ubiquitinating and inhibiting the class II PI3K-C2βXinjiang Cai, Shekhar Srivastava, Yi Sun, et al.Journal of Cell Science|September 15, 2007
Cdc42 is required for EGF-stimulated protrusion and motility in MTLn3 carcinoma cellsMirvat El-Sibai, Peri Nalbant, Huan Pang, et al.Pageof 13