Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Gilberto Fronza

Showing results (21-30 of 65) with videos related to

Pageof 7
Sort By:
Mutagenesis|October 20, 2007
Transcriptional properties of feline p53 and its tumour-associated mutants: a yeast-based approachUmberto Cardellino, Yari Ciribilli, Virginia Andreotti, et al.
Oncogene|March 16, 2002
Tumour p53 mutations exhibit promoter selective dominance over wild type p53Paola Monti, Paola Campomenosi, Yari Ciribilli, et al.
DNA Repair|June 17, 2011
3-Methyl-3-deazaadenine, a stable isostere of N3-methyl-adenine, is efficiently bypassed by replication in vivo and by transcription in vitroPaola Monti, Christopher Broxson, Alberto Inga, et al.
Journal of Cellular Biochemistry|April 7, 2006
Stable formation of mutated p53 multimers in a Chinese hamster cell line causes defective p53 nuclear localization and abrogates its residual functionLaura Ottaggio, Paola Campomenosi, Gilberto Fronza, et al.
Bioscience Reports|February 13, 2019
Autophagy induced by SAHA affects mutant P53 degradation and cancer cell survivalGiorgia Foggetti, Laura Ottaggio, Debora Russo, et al.
Genes|March 6, 2021
Evaluating the Influence of a G-Quadruplex Prone Sequence on the Transactivation Potential by Wild-Type and/or Mutant P53 Family Proteins through a Yeast-Based Functional AssayPaola Monti, Vaclav Brazda, Natália Bohálová, et al.
Oncotarget|June 14, 2014
∆N-P63α and TA-P63α exhibit intrinsic differences in transactivation specificities that depend on distinct features of DNA target sitesPaola Monti, Yari Ciribilli, Alessandra Bisio, et al.
DNA Repair|May 18, 2010
XRCC1 deficiency influences the cytotoxicity and the genomic instability induced by Me-lex, a specific inducer of N3-methyladenineDebora Russo, Gilberto Fronza, Laura Ottaggio, et al.
Biochemistry|May 12, 2004
Nucleotide excision repair defect influences lethality and mutagenicity induced by Me-lex, a sequence-selective N3-adenine methylating agent in the absence of base excision repairPaola Monti, Raffaella Iannone, Paola Campomenosi, et al.
Experimental Cell Research|September 30, 2014
Studying p53 family proteins in yeast: induction of autophagic cell death and modulation by interactors and small moleculesMariana Leão, Sara Gomes, Cláudia Bessa, et al.
Pageof 7

Showing results (21-30 of 65) with videos related to

Sort By:
Pageof 7
Mutagenesis|October 20, 2007
Transcriptional properties of feline p53 and its tumour-associated mutants: a yeast-based approachUmberto Cardellino, Yari Ciribilli, Virginia Andreotti, et al.
Oncogene|March 16, 2002
Tumour p53 mutations exhibit promoter selective dominance over wild type p53Paola Monti, Paola Campomenosi, Yari Ciribilli, et al.
DNA Repair|June 17, 2011
3-Methyl-3-deazaadenine, a stable isostere of N3-methyl-adenine, is efficiently bypassed by replication in vivo and by transcription in vitroPaola Monti, Christopher Broxson, Alberto Inga, et al.
Journal of Cellular Biochemistry|April 7, 2006
Stable formation of mutated p53 multimers in a Chinese hamster cell line causes defective p53 nuclear localization and abrogates its residual functionLaura Ottaggio, Paola Campomenosi, Gilberto Fronza, et al.
Bioscience Reports|February 13, 2019
Autophagy induced by SAHA affects mutant P53 degradation and cancer cell survivalGiorgia Foggetti, Laura Ottaggio, Debora Russo, et al.
Genes|March 6, 2021
Evaluating the Influence of a G-Quadruplex Prone Sequence on the Transactivation Potential by Wild-Type and/or Mutant P53 Family Proteins through a Yeast-Based Functional AssayPaola Monti, Vaclav Brazda, Natália Bohálová, et al.
Oncotarget|June 14, 2014
∆N-P63α and TA-P63α exhibit intrinsic differences in transactivation specificities that depend on distinct features of DNA target sitesPaola Monti, Yari Ciribilli, Alessandra Bisio, et al.
DNA Repair|May 18, 2010
XRCC1 deficiency influences the cytotoxicity and the genomic instability induced by Me-lex, a specific inducer of N3-methyladenineDebora Russo, Gilberto Fronza, Laura Ottaggio, et al.
Biochemistry|May 12, 2004
Nucleotide excision repair defect influences lethality and mutagenicity induced by Me-lex, a sequence-selective N3-adenine methylating agent in the absence of base excision repairPaola Monti, Raffaella Iannone, Paola Campomenosi, et al.
Experimental Cell Research|September 30, 2014
Studying p53 family proteins in yeast: induction of autophagic cell death and modulation by interactors and small moleculesMariana Leão, Sara Gomes, Cláudia Bessa, et al.
Pageof 7