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Cell Reports|December 12, 2019
Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's DiseaseEleanor K Pickett, Abigail G Herrmann, Jamie McQueen, et al.Translational Psychiatry|February 20, 2021
Functional brain defects in a mouse model of a chromosomal t(1;11) translocation that disrupts DISC1 and confers increased risk of psychiatric illnessMarion Bonneau, Shane T O' Sullivan, Miguel A Gonzalez-Lozano, et al.Nature Neuroscience|January 6, 2025
Typical development of synaptic and neuronal properties can proceed without microglia in the cortex and thalamusMary O'Keeffe, Sam A Booker, Darren Walsh, et al.Nature Communications|January 11, 2022
Reactive astrocytes acquire neuroprotective as well as deleterious signatures in response to Tau and Aß pathologyZoeb Jiwaji, Sachin S Tiwari, Rolando X Avilés-Reyes, et al.Nature Communications|July 21, 2019
Deletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populationsRocío Rojo, Anna Raper, Derya D Ozdemir, et al.Nature Communications|January 26, 2018
C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicityBhuvaneish T Selvaraj, Matthew R Livesey, Chen Zhao, et al.Cell Death and Differentiation|December 13, 2017
Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseasesNiamh M C Connolly, Pierre Theurey, Vera Adam-Vizi, et al.Neuron|November 18, 2017
Building Bridges through ScienceThomas Lissek, Michelle Adams, John Adelman, et al.Pageof 21