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Giles E Hardingham

Showing results (111-120 of 124) with videos related to

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Nature Communications|June 8, 2023
Astrocyte-oligodendrocyte interaction regulates central nervous system regenerationIrene Molina-Gonzalez, Rebecca K Holloway, Zoeb Jiwaji, et al.
Molecular Psychiatry|November 8, 2018
Reversal of proliferation deficits caused by chromosome 16p13.11 microduplication through targeting NFκB signaling: an integrated study of patient-derived neuronal precursor cells, cerebral organoids and in vivo brain imagingMandy Johnstone, Navneet A Vasistha, Miruna C Barbu, et al.
Brain Communications|March 30, 2022
Transactive response DNA-binding protein-43 proteinopathy in oligodendrocytes revealed using an induced pluripotent stem cell modelSamantha K Barton, Dario Magnani, Owen G James, et al.
Brain : a Journal of Neurology|December 14, 2018
GRIN2A-related disorders: genotype and functional consequence predict phenotypeVincent Strehlow, Henrike O Heyne, Danique R M Vlaskamp, et al.
Journal of Neurochemistry|July 21, 2025
Embracing the Modern Biochemistry of Brain MetabolismJuan P Bolaños, Cristina M Alberini, Angeles Almeida, et al.
Molecular Psychiatry|September 5, 2019
Familial t(1;11) translocation is associated with disruption of white matter structural integrity and oligodendrocyte-myelin dysfunctionNavneet A Vasistha, Mandy Johnstone, Samantha K Barton, et al.
Cell Reports. Medicine|August 31, 2023
Human astrocytes and microglia show augmented ingestion of synapses in Alzheimer's disease via MFG-E8Makis Tzioras, Michael J D Daniels, Caitlin Davies, et al.
Cell Reports|December 12, 2019
Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's DiseaseEleanor K Pickett, Abigail G Herrmann, Jamie McQueen, et al.
Translational Psychiatry|February 20, 2021
Functional brain defects in a mouse model of a chromosomal t(1;11) translocation that disrupts DISC1 and confers increased risk of psychiatric illnessMarion Bonneau, Shane T O' Sullivan, Miguel A Gonzalez-Lozano, et al.
Nature Neuroscience|January 6, 2025
Typical development of synaptic and neuronal properties can proceed without microglia in the cortex and thalamusMary O'Keeffe, Sam A Booker, Darren Walsh, et al.
Pageof 13

Showing results (111-120 of 124) with videos related to

Sort By:
Pageof 13
Nature Communications|June 8, 2023
Astrocyte-oligodendrocyte interaction regulates central nervous system regenerationIrene Molina-Gonzalez, Rebecca K Holloway, Zoeb Jiwaji, et al.
Molecular Psychiatry|November 8, 2018
Reversal of proliferation deficits caused by chromosome 16p13.11 microduplication through targeting NFκB signaling: an integrated study of patient-derived neuronal precursor cells, cerebral organoids and in vivo brain imagingMandy Johnstone, Navneet A Vasistha, Miruna C Barbu, et al.
Brain Communications|March 30, 2022
Transactive response DNA-binding protein-43 proteinopathy in oligodendrocytes revealed using an induced pluripotent stem cell modelSamantha K Barton, Dario Magnani, Owen G James, et al.
Brain : a Journal of Neurology|December 14, 2018
GRIN2A-related disorders: genotype and functional consequence predict phenotypeVincent Strehlow, Henrike O Heyne, Danique R M Vlaskamp, et al.
Journal of Neurochemistry|July 21, 2025
Embracing the Modern Biochemistry of Brain MetabolismJuan P Bolaños, Cristina M Alberini, Angeles Almeida, et al.
Molecular Psychiatry|September 5, 2019
Familial t(1;11) translocation is associated with disruption of white matter structural integrity and oligodendrocyte-myelin dysfunctionNavneet A Vasistha, Mandy Johnstone, Samantha K Barton, et al.
Cell Reports. Medicine|August 31, 2023
Human astrocytes and microglia show augmented ingestion of synapses in Alzheimer's disease via MFG-E8Makis Tzioras, Michael J D Daniels, Caitlin Davies, et al.
Cell Reports|December 12, 2019
Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's DiseaseEleanor K Pickett, Abigail G Herrmann, Jamie McQueen, et al.
Translational Psychiatry|February 20, 2021
Functional brain defects in a mouse model of a chromosomal t(1;11) translocation that disrupts DISC1 and confers increased risk of psychiatric illnessMarion Bonneau, Shane T O' Sullivan, Miguel A Gonzalez-Lozano, et al.
Nature Neuroscience|January 6, 2025
Typical development of synaptic and neuronal properties can proceed without microglia in the cortex and thalamusMary O'Keeffe, Sam A Booker, Darren Walsh, et al.
Pageof 13