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The American Journal of Gastroenterology|August 26, 2025
TIMING THE SCAN: OPTIMIZING SCREENING FOR OSTEOPOROSIS AND RISK OF FRACTURE IN CELIAC DISEASEFrancesco Tovoli, Guido Zavatta, Giovanni Monaco, et al.
Cold Spring Harbor Perspectives in Biology|September 11, 2019
Bcl-2-Protein Family as Modulators of IP3 Receptors and Other Organellar Ca2+ ChannelsHristina Ivanova, Tim Vervliet, Giovanni Monaco, et al.
FEBS Letters|July 4, 2020
Small molecule 3PO inhibits glycolysis but does not bind to 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase-3 (PFKFB3)Besa Emini Veseli, Paola Perrotta, Pieter Van Wielendaele, et al.
The FEBS Journal|November 14, 2017
A double point mutation at residues Ile14 and Val15 of Bcl-2 uncovers a role for the BH4 domain in both protein stability and functionGiovanni Monaco, Rita La Rovere, Spyridoula Karamanou, et al.
The Journal of Biological Chemistry|February 15, 2015
The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondriaGiovanni Monaco, Elke Decrock, Nir Arbel, et al.
Oncotarget|August 6, 2016
The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibitionHristina Ivanova, Abigael Ritaine, Larry Wagner, et al.
Blood|January 4, 2011
Induction of Ca²+-driven apoptosis in chronic lymphocytic leukemia cells by peptide-mediated disruption of Bcl-2-IP3 receptor interactionFei Zhong, Michael W Harr, Geert Bultynck, et al.
Plos One|October 19, 2013
Alpha-helical destabilization of the Bcl-2-BH4-domain peptide abolishes its ability to inhibit the IP3 receptorGiovanni Monaco, Elke Decrock, Koen Nuyts, et al.
Scientific Reports|April 16, 2015
Ryanodine receptors are targeted by anti-apoptotic Bcl-XL involving its BH4 domain and Lys87 from its BH3 domainTim Vervliet, Irma Lemmens, Elien Vandermarliere, et al.
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