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EMBO Reports|April 20, 2017
Posttranslational modification impact on the mechanism by which amyloid-β induces synaptic dysfunctionKatarzyna M Grochowska, PingAn Yuanxiang, Julia Bär, et al.Molecular Pharmacology|May 4, 2019
Preferred Formation of Heteromeric Channels between Coexpressed SK1 and IKCa Channel Subunits Provides a Unique Pharmacological Profile of Ca2+-Activated Potassium ChannelsJames Higham, Giriraj Sahu, Rima-Marie Wazen, et al.Molecular Brain|August 13, 2017
A T-type channel-calmodulin complex triggers αCaMKII activationHadhimulya Asmara, Ileana Micu, Arsalan P Rizwan, et al.Angiogenesis|December 17, 2014
Tipping off endothelial tubes: nitric oxide drives tip cellsMani Krishna Priya, Giriraj Sahu, David R Soto-Pantoja, et al.Journal of Cell Science|February 24, 2012
Molecular interactions of the physiological anti-hypertensive peptide catestatin with the neuronal nicotinic acetylcholine receptorBhavani S Sahu, Jagan Mohan, Jagan M Obbineni, et al.The Journal of Biological Chemistry|October 30, 2012
Functional genetic variants of the catecholamine-release-inhibitory peptide catestatin in an Indian population: allele-specific effects on metabolic traitsBhavani S Sahu, Jagan M Obbineni, Giriraj Sahu, et al.Neuron|December 18, 2025
Elevated calneuron-1, an accessory subunit of muscarinic receptors, induces frontotemporal dysconnectivity and schizophrenia-like deficitsAnja M Oelschlegel, Jastyn A Pöpplau, Alexandre Ryzynski, et al.Pageof 2