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Giulia Petroni

Showing results (71-80 of 79) with videos related to

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The Journal of Allergy and Clinical Immunology|March 19, 2013
A novel allergen-adjuvant conjugate suitable for specific immunotherapy of respiratory allergyLucia Filì, Alessandra Vultaggio, Elisa Cardilicchia, et al.
Cell Death & Disease|December 22, 2025
Th17-like cells and immunosuppressive macrophages infiltrate tertiary lymphoid structures with distinct maturation status in soft-tissue sarcomaGiulia Petroni, Federico Scolari, Guido Scoccianti, et al.
Translational Oncology|February 28, 2020
hERG1 and HIF-2α Behave as Biomarkers of Positive Response to Bevacizumab in Metastatic Colorectal Cancer PatientsJessica Iorio, Elena Lastraioli, Lorenzo Tofani, et al.
Nature Immunology|August 5, 2020
Mitochondrial DNA drives abscopal responses to radiation that are inhibited by autophagyTakahiro Yamazaki, Alexander Kirchmair, Ai Sato, et al.
Science Signaling|April 6, 2017
The conformational state of hERG1 channels determines integrin association, downstream signaling, and cancer progressionAndrea Becchetti, Silvia Crescioli, Francesca Zanieri, et al.
Journal of the National Cancer Institute|December 11, 2024
Impact of radiation therapy dose, fractionation, and immunotherapeutic partner in a mouse model of hormone receptor-positive mammary carcinogenesisAitziber Buqué, Norma Bloy, Giulia Petroni, et al.
Nature Cancer|July 7, 2025
IL-17A-secreting γδ T cells promote resistance to CDK4/CDK6 inhibitors in HR<sup>+</sup>HER2<sup>-</sup> breast cancer via CX3CR1<sup>+</sup> macrophagesGiulia Petroni, Claudia Galassi, Kenneth H Gouin, et al.
Nature Cancer|July 23, 2025
Author Correction: IL-17A-secreting γδ T cells promote resistance to CDK4/CDK6 inhibitors in HR<sup>+</sup>HER2<sup>-</sup> breast cancer via CX3CR1<sup>+</sup> macrophagesGiulia Petroni, Claudia Galassi, Kenneth H Gouin, et al.
The EMBO Journal|August 30, 2021
Autophagy in major human diseasesDaniel J Klionsky, Giulia Petroni, Ravi K Amaravadi, et al.
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Showing results (71-80 of 79) with videos related to

Sort By:
Pageof 8
You have reached the last page of results.This site can display upto 79 results.
The Journal of Allergy and Clinical Immunology|March 19, 2013
A novel allergen-adjuvant conjugate suitable for specific immunotherapy of respiratory allergyLucia Filì, Alessandra Vultaggio, Elisa Cardilicchia, et al.
Cell Death & Disease|December 22, 2025
Th17-like cells and immunosuppressive macrophages infiltrate tertiary lymphoid structures with distinct maturation status in soft-tissue sarcomaGiulia Petroni, Federico Scolari, Guido Scoccianti, et al.
Translational Oncology|February 28, 2020
hERG1 and HIF-2α Behave as Biomarkers of Positive Response to Bevacizumab in Metastatic Colorectal Cancer PatientsJessica Iorio, Elena Lastraioli, Lorenzo Tofani, et al.
Nature Immunology|August 5, 2020
Mitochondrial DNA drives abscopal responses to radiation that are inhibited by autophagyTakahiro Yamazaki, Alexander Kirchmair, Ai Sato, et al.
Science Signaling|April 6, 2017
The conformational state of hERG1 channels determines integrin association, downstream signaling, and cancer progressionAndrea Becchetti, Silvia Crescioli, Francesca Zanieri, et al.
Journal of the National Cancer Institute|December 11, 2024
Impact of radiation therapy dose, fractionation, and immunotherapeutic partner in a mouse model of hormone receptor-positive mammary carcinogenesisAitziber Buqué, Norma Bloy, Giulia Petroni, et al.
Nature Cancer|July 7, 2025
IL-17A-secreting γδ T cells promote resistance to CDK4/CDK6 inhibitors in HR<sup>+</sup>HER2<sup>-</sup> breast cancer via CX3CR1<sup>+</sup> macrophagesGiulia Petroni, Claudia Galassi, Kenneth H Gouin, et al.
Nature Cancer|July 23, 2025
Author Correction: IL-17A-secreting γδ T cells promote resistance to CDK4/CDK6 inhibitors in HR<sup>+</sup>HER2<sup>-</sup> breast cancer via CX3CR1<sup>+</sup> macrophagesGiulia Petroni, Claudia Galassi, Kenneth H Gouin, et al.
The EMBO Journal|August 30, 2021
Autophagy in major human diseasesDaniel J Klionsky, Giulia Petroni, Ravi K Amaravadi, et al.
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