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Biochemical Pharmacology|February 21, 2012
p38(MAPK) and ERK1/2 dictate cell death/survival response to different pro-oxidant stimuli via p53 and Nrf2 in neuroblastoma cells SH-SY5YGiuseppe Filomeni, Sara Piccirillo, Giuseppe Rotilio, et al.Antioxidants & Redox Signaling|February 12, 2005
Antiapoptotic response to induced GSH depletion: involvement of heat shock proteins and NF-kappaB activationGiuseppe Filomeni, Katia Aquilano, Giuseppe Rotilio, et al.Molecular Pharmacology|August 5, 2008
Reactive oxygen species mediate p53 activation and apoptosis induced by sodium nitroprusside in SH-SY5Y cellsSimone Cardaci, Giuseppe Filomeni, Giuseppe Rotilio, et al.Cancer Research|December 17, 2005
Glutathione-related systems and modulation of extracellular signal-regulated kinases are involved in the resistance of AGS adenocarcinoma gastric cells to diallyl disulfide-induced apoptosisGiuseppe Filomeni, Katia Aquilano, Giuseppe Rotilio, et al.The Biochemical Journal|July 2, 2010
p38(MAPK)/p53 signalling axis mediates neuronal apoptosis in response to tetrahydrobiopterin-induced oxidative stress and glucose uptake inhibition: implication for neurodegenerationSimone Cardaci, Giuseppe Filomeni, Giuseppe Rotilio, et al.Autophagy|July 20, 2010
Under the ROS…thiol network is the principal suspect for autophagy commitmentGiuseppe Filomeni, Enrico Desideri, Simone Cardaci, et al.International Journal of Cell Biology|March 15, 2014
S-nitrosation and ubiquitin-proteasome system interplay in neuromuscular disordersSalvatore Rizza, Costanza Montagna, Giuseppina Di Giacomo, et al.Journal of Cellular and Molecular Medicine|December 2, 2008
Tau dephosphorylation and microfilaments disruption are upstream events of the anti-proliferative effects of DADS in SH-SY5Y cellsKatia Aquilano, Paola Vigilanza, Giuseppe Filomeni, et al.Free Radical Biology & Medicine|July 5, 2005
Activation of c-Jun-N-terminal kinase is required for apoptosis triggered by glutathione disulfide in neuroblastoma cellsGiuseppe Filomeni, Katia Aquilano, Patrizia Civitareale, et al.The Journal of Biological Chemistry|August 1, 2009
Redox mechanisms involved in the selective activation of Nrf2-mediated resistance versus p53-dependent apoptosis in adenocarcinoma cellsSara Piccirillo, Giuseppe Filomeni, Bernhard Brüne, et al.Pageof 9