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Biochemical Pharmacology|February 24, 2020
S-nitrosylation affects TRAP1 structure and ATPase activity and modulates cell response to apoptotic stimuliFiorella Faienza, Matteo Lambrughi, Salvatore Rizza, et al.BMC Cancer|November 13, 2007
PYRROLO[1,2-b][1,2,5]BENZOTHIADIAZEPINES (PBTDs) induce apoptosis in K562 cellsGabriella Marfe, Carla Di Stefano, Romano Silvestri, et al.Redox Biology|May 27, 2025
Disulfide-mediated tetramerization of TRAP1 fosters its antioxidant and pro-neoplastic activitiesFiorella Faienza, Claudio Laquatra, Matteo Castelli, et al.EMBO Reports|November 27, 2020
Redox activation of ATM enhances GSNOR translation to sustain mitophagy and tolerance to oxidative stressClaudia Cirotti, Salvatore Rizza, Paola Giglio, et al.Antioxidants & Redox Signaling|April 2, 2014
S-nitrosoglutathione reductase deficiency-induced S-nitrosylation results in neuromuscular dysfunctionCostanza Montagna, Giuseppina Di Giacomo, Salvatore Rizza, et al.Cell Reports|January 19, 2023
GSNOR deficiency promotes tumor growth via FAK1 S-nitrosylationSalvatore Rizza, Luca Di Leo, Chiara Pecorari, et al.Journal of Medicinal Chemistry|September 15, 2006
Pyrrolo[1,2-b][1,2,5]benzothiadiazepines (PBTDs): A new class of agents with high apoptotic activity in chronic myelogenous leukemia K562 cells and in cells from patients at onset and who were imatinib-resistantRomano Silvestri, Gabriella Marfè, Marino Artico, et al.Proceedings of the National Academy of Sciences of the United States of America|June 13, 2024
AMBRA1 levels predict resistance to MAPK inhibitors in melanomaLuca Di Leo, Chiara Pagliuca, Ali Kishk, et al.Cellular and Molecular Life Sciences : CMLS|August 16, 2023
AMBRA1 phosphorylation by CDK1 and PLK1 regulates mitotic spindle orientationFiorella Faienza, Federica Polverino, Girish Rajendraprasad, et al.Nature Communications|May 6, 2021
Loss of Ambra1 promotes melanoma growth and invasionLuca Di Leo, Valérie Bodemeyer, Francesca M Bosisio, et al.Pageof 9