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Toxicology and Applied Pharmacology|October 14, 2003
Use of Hsf1(-/-) mice reveals an essential role for HSF1 to protect lung against cadmium-induced injuryDelphine Wirth, Elisabeth Christians, Xiang Li, et al.Antioxidants & Redox Signaling|February 12, 2005
Mouse HSF1 disruption perturbs redox state and increases mitochondrial oxidative stress in kidneyLiang-Jun Yan, Namakkal S Rajasekaran, Srinivasan Sathyanarayanan, et al.American Journal of Physiology. Heart and Circulatory Physiology|July 10, 2018
Aging-related increase in store-operated Ca<sup>2+</sup> influx in human ventricular fibroblastsMomin Mohis, Stacie Edwards, Sean Ryan, et al.Genes & Development|June 5, 2003
Localized recruitment of a chromatin-remodeling activity by an activator in vivo drives transcriptional elongationLaura L Corey, Christine S Weirich, Ivor J Benjamin, et al.The Journal of Pharmacology and Experimental Therapeutics|December 2, 2011
The role of β-arrestin2 in the mechanism of morphine tolerance in the mouse and guinea pig gastrointestinal tractMinho Kang, Hercules T Maguma, Tricia H Smith, et al.American Journal of Hypertension|April 19, 2025
Machine Learning and Artificial Intelligence for Research on HypertensionFatima Zohra Khamissi, Lorelle Sun, Paige Johnson, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 12, 2007
Unmasking different mechanical and energetic roles for the small heat shock proteins CryAB and HSPB2 using genetically modified mouse heartsIlka Pinz, Jeffrey Robbins, Namakkal S Rajasekaran, et al.Endocrinology|July 16, 2010
Adrenomedullin relaxes rat uterine artery: mechanisms and influence of pregnancy and estradiolGracious R Ross, Uma Yallampalli, Pandu R R Gangula, et al.Genes & Development|February 5, 2008
Differential display of DNA-binding proteins reveals heat-shock factor 1 as a circadian transcription factorHans Reinke, Camille Saini, Fabienne Fleury-Olela, et al.Journal of Molecular and Cellular Cardiology|September 25, 2010
Selective degradation of aggregate-prone CryAB mutants by HSPB1 is mediated by ubiquitin-proteasome pathwaysHuali Zhang, Namakkal S Rajasekaran, Andras Orosz, et al.Pageof 11