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Science Translational Medicine|December 8, 2022
The path to venetoclax resistance is paved with mutations, metabolism, and moreGraeme P Sullivan, Lyndsey Flanagan, Daniel Alencar Rodrigues, et al.The FEBS Journal|April 5, 2017
Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokinesDanielle M Clancy, Conor M Henry, Graeme P Sullivan, et al.Frontiers in Immunology|July 18, 2022
IL-1α and IL-36 Family Cytokines Can Undergo Processing and Activation by Diverse Allergen-Associated ProteasesValentina Frezza, Zaneta Najda, Pavel Davidovich, et al.Chemical Communications (Cambridge, England)|October 4, 2023
First-in-class metallo-PROTAC as an effective degrader of select Pt-binding proteinsPaul D O'Dowd, Graeme P Sullivan, Daniel A Rodrigues, et al.Cell Reports|January 19, 2016
Neutrophil-Derived Proteases Escalate Inflammation through Activation of IL-36 Family CytokinesConor M Henry, Graeme P Sullivan, Danielle M Clancy, et al.FEBS Open Bio|May 31, 2016
Production of biologically active IL-36 family cytokines through insertion of N-terminal caspase cleavage motifsDanielle M Clancy, Conor M Henry, Pavel B Davidovich, et al.Developmental Cell|February 29, 2020
TRAIL Receptors Serve as Stress-Associated Molecular Patterns to Promote ER-Stress-Induced InflammationGraeme P Sullivan, Hazel O'Connor, Conor M Henry, et al.Cell Death & Disease|March 9, 2018
Suppressing IL-36-driven inflammation using peptide pseudosubstrates for neutrophil proteasesGraeme P Sullivan, Conor M Henry, Danielle M Clancy, et al.Cell Reports|March 15, 2018
Extracellular Neutrophil Proteases Are Efficient Regulators of IL-1, IL-33, and IL-36 Cytokine Activity but Poor Effectors of Microbial KillingDanielle M Clancy, Graeme P Sullivan, Hannah B T Moran, et al.FEBS Open Bio|May 11, 2018
Identification of small-molecule elastase inhibitors as antagonists of IL-36 cytokine activationGraeme P Sullivan, Pavel B Davidovich, Sylvia Sura-Trueba, et al.Pageof 2