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Grant Dewson

Showing results (41-50 of 79) with videos related to

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Elife|February 10, 2017
Disordered clusters of Bak dimers rupture mitochondria during apoptosisRachel T Uren, Martin O'Hely, Sweta Iyer, et al.
The Journal of Biological Chemistry|September 21, 2010
Inhibition of Bak activation by VDAC2 is dependent on the Bak transmembrane anchorMichael Lazarou, Diana Stojanovski, Ann E Frazier, et al.
Structure (London, England : 1993)|October 18, 2011
Crystal structure of a BCL-W domain-swapped dimer: implications for the function of BCL-2 family proteinsErinna F Lee, Grant Dewson, Brian J Smith, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 5, 2017
BAK α6 permits activation by BH3-only proteins and homooligomerization via the canonical hydrophobic grooveMark Xiang Li, Iris K L Tan, Stephen B Ma, et al.
Cell Reports|April 11, 2019
BAX Activation: Mutations Near Its Proposed Non-canonical BH3 Binding Site Reveal Allosteric Changes Controlling Mitochondrial AssociationMichael A Dengler, Adeline Y Robin, Leonie Gibson, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 18, 2014
Apoptotic pore formation is associated with in-plane insertion of Bak or Bax central helices into the mitochondrial outer membraneDana Westphal, Grant Dewson, Marie Menard, et al.
Nature Communications|May 25, 2016
Identification of an activation site in Bak and mitochondrial Bax triggered by antibodiesSweta Iyer, Khatira Anwari, Amber E Alsop, et al.
Molecular Cell|September 2, 2014
Bak core and latch domains separate during activation, and freed core domains form symmetric homodimersJason M Brouwer, Dana Westphal, Grant Dewson, et al.
Nature|December 21, 2021
Activation mechanism of PINK1Zhong Yan Gan, Sylvie Callegari, Simon A Cobbold, et al.
Cell Death and Differentiation|September 28, 2022
Mitochondrial E3 ubiquitin ligase MARCHF5 controls BAK apoptotic activity independently of BH3-only proteinsAllan Shuai Huang, Hui San Chin, Boris Reljic, et al.
Pageof 8

Showing results (41-50 of 79) with videos related to

Sort By:
Pageof 8
Elife|February 10, 2017
Disordered clusters of Bak dimers rupture mitochondria during apoptosisRachel T Uren, Martin O'Hely, Sweta Iyer, et al.
The Journal of Biological Chemistry|September 21, 2010
Inhibition of Bak activation by VDAC2 is dependent on the Bak transmembrane anchorMichael Lazarou, Diana Stojanovski, Ann E Frazier, et al.
Structure (London, England : 1993)|October 18, 2011
Crystal structure of a BCL-W domain-swapped dimer: implications for the function of BCL-2 family proteinsErinna F Lee, Grant Dewson, Brian J Smith, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 5, 2017
BAK α6 permits activation by BH3-only proteins and homooligomerization via the canonical hydrophobic grooveMark Xiang Li, Iris K L Tan, Stephen B Ma, et al.
Cell Reports|April 11, 2019
BAX Activation: Mutations Near Its Proposed Non-canonical BH3 Binding Site Reveal Allosteric Changes Controlling Mitochondrial AssociationMichael A Dengler, Adeline Y Robin, Leonie Gibson, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 18, 2014
Apoptotic pore formation is associated with in-plane insertion of Bak or Bax central helices into the mitochondrial outer membraneDana Westphal, Grant Dewson, Marie Menard, et al.
Nature Communications|May 25, 2016
Identification of an activation site in Bak and mitochondrial Bax triggered by antibodiesSweta Iyer, Khatira Anwari, Amber E Alsop, et al.
Molecular Cell|September 2, 2014
Bak core and latch domains separate during activation, and freed core domains form symmetric homodimersJason M Brouwer, Dana Westphal, Grant Dewson, et al.
Nature|December 21, 2021
Activation mechanism of PINK1Zhong Yan Gan, Sylvie Callegari, Simon A Cobbold, et al.
Cell Death and Differentiation|September 28, 2022
Mitochondrial E3 ubiquitin ligase MARCHF5 controls BAK apoptotic activity independently of BH3-only proteinsAllan Shuai Huang, Hui San Chin, Boris Reljic, et al.
Pageof 8