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JAAD Case Reports
|
January 22, 2025
High-grade sarcoma arising within a cellular dermatofibroma
Nelson Ugwu, Nina Ran, Grant Fischer, et al.
Biorxiv : the Preprint Server for Biology
|
August 2, 2024
PRAME expression in melanoma is negatively regulated by TET2-mediated DNA hydroxymethylation
Rui Fang, Tuulia Vallius, Arianna Zhang, et al.
Journal for Immunotherapy of Cancer
|
June 26, 2020
Combination treatment with radiotherapy and a novel oxidative phosphorylation inhibitor overcomes PD-1 resistance and enhances antitumor immunity
Dawei Chen, Hampartsoum B Barsoumian, Grant Fischer, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology
|
March 2, 2025
PRAME Expression in Melanoma is Negatively Regulated by TET2-Mediated DNA Hydroxymethylation
Rui Fang, Tuulia Vallius, Arianna Zhang, et al.
Human Molecular Genetics
|
April 18, 2013
Smooth muscle hyperplasia due to loss of smooth muscle α-actin is driven by activation of focal adhesion kinase, altered p53 localization and increased levels of platelet-derived growth factor receptor-β
Christina L Papke, Jiumei Cao, Callie S Kwartler, et al.
Page
of 1
Search research articles
Search
Showing results (1-10 of 5) with videos related to
Sort By:
Page
of 1
JAAD Case Reports
|
January 22, 2025
High-grade sarcoma arising within a cellular dermatofibroma
Nelson Ugwu, Nina Ran, Grant Fischer, et al.
Biorxiv : the Preprint Server for Biology
|
August 2, 2024
PRAME expression in melanoma is negatively regulated by TET2-mediated DNA hydroxymethylation
Rui Fang, Tuulia Vallius, Arianna Zhang, et al.
Journal for Immunotherapy of Cancer
|
June 26, 2020
Combination treatment with radiotherapy and a novel oxidative phosphorylation inhibitor overcomes PD-1 resistance and enhances antitumor immunity
Dawei Chen, Hampartsoum B Barsoumian, Grant Fischer, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology
|
March 2, 2025
PRAME Expression in Melanoma is Negatively Regulated by TET2-Mediated DNA Hydroxymethylation
Rui Fang, Tuulia Vallius, Arianna Zhang, et al.
Human Molecular Genetics
|
April 18, 2013
Smooth muscle hyperplasia due to loss of smooth muscle α-actin is driven by activation of focal adhesion kinase, altered p53 localization and increased levels of platelet-derived growth factor receptor-β
Christina L Papke, Jiumei Cao, Callie S Kwartler, et al.
Page
of 1