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Gretl Hendrickx

Showing results (1-10 of 23) with videos related to

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Nature Reviews. Rheumatology|April 23, 2015
A look behind the scenes: the risk and pathogenesis of primary osteoporosisGretl Hendrickx, Eveline Boudin, Wim Van Hul
Nature Reviews. Endocrinology|May 20, 2026
The pathogenesis and intersecting mechanisms of monogenic bone mass disordersRaja Padidela, Gretl Hendrickx, Geert Mortier, et al.
Molecular and Cellular Endocrinology|January 10, 2016
Genetic control of bone massEveline Boudin, Igor Fijalkowski, Gretl Hendrickx, et al.
Calcified Tissue International|November 25, 2019
WNT16 Requires Gα Subunits as Intracellular Partners for Both Its Canonical and Non-Canonical WNT Signalling Activity in OsteoblastsGretl Hendrickx, Eveline Boudin, Marinus Verbeek, et al.
Clinical Genetics|December 18, 2023
RUNX2-related metaphyseal dysplasia with maxillary hypoplasia: A rare skeletal disorder resembling SFRP4-related Pyle diseaseEwa Hordyjewska-Kowalczyk, Wim Wuyts, Nele Boeckx, et al.
Calcified Tissue International|January 13, 2017
Genetic Screening of WNT4 and WNT5B in Two Populations with Deviating Bone Mineral DensitiesGretl Hendrickx, Eveline Boudin, Ellen Steenackers, et al.
Bone|November 5, 2013
Variation in the Kozak sequence of WNT16 results in an increased translation and is associated with osteoporosis related parametersGretl Hendrickx, Eveline Boudin, Igor Fijałkowski, et al.
Calcified Tissue International|September 20, 2023
Genetic Screening of ZNF687 and PFN1 in a Paget's Disease of Bone Cohort Indicates an Important Role for the Nuclear Localization Signal of ZNF687Yentl Huybrechts, Raphaël De Ridder, Ellen Steenackers, et al.
The Journal of Clinical Endocrinology and Metabolism|January 4, 2024
A Mosaic Variant in CTNNB1/β-catenin as a Novel Cause for Osteopathia Striata With Cranial SclerosisYentl Huybrechts, Natasha M Appelman-Dijkstra, Ellen Steenackers, et al.
Calcified Tissue International|December 5, 2023
An Additional Lrp4 High Bone Mass Mutation Mitigates the Sost-Knockout Phenotype in Mice by Increasing Bone RemodelingGretl Hendrickx, Eveline Boudin, Ligia Mateiu, et al.
Pageof 3

Showing results (1-10 of 23) with videos related to

Sort By:
Pageof 3
Nature Reviews. Rheumatology|April 23, 2015
A look behind the scenes: the risk and pathogenesis of primary osteoporosisGretl Hendrickx, Eveline Boudin, Wim Van Hul
Nature Reviews. Endocrinology|May 20, 2026
The pathogenesis and intersecting mechanisms of monogenic bone mass disordersRaja Padidela, Gretl Hendrickx, Geert Mortier, et al.
Molecular and Cellular Endocrinology|January 10, 2016
Genetic control of bone massEveline Boudin, Igor Fijalkowski, Gretl Hendrickx, et al.
Calcified Tissue International|November 25, 2019
WNT16 Requires Gα Subunits as Intracellular Partners for Both Its Canonical and Non-Canonical WNT Signalling Activity in OsteoblastsGretl Hendrickx, Eveline Boudin, Marinus Verbeek, et al.
Clinical Genetics|December 18, 2023
RUNX2-related metaphyseal dysplasia with maxillary hypoplasia: A rare skeletal disorder resembling SFRP4-related Pyle diseaseEwa Hordyjewska-Kowalczyk, Wim Wuyts, Nele Boeckx, et al.
Calcified Tissue International|January 13, 2017
Genetic Screening of WNT4 and WNT5B in Two Populations with Deviating Bone Mineral DensitiesGretl Hendrickx, Eveline Boudin, Ellen Steenackers, et al.
Bone|November 5, 2013
Variation in the Kozak sequence of WNT16 results in an increased translation and is associated with osteoporosis related parametersGretl Hendrickx, Eveline Boudin, Igor Fijałkowski, et al.
Calcified Tissue International|September 20, 2023
Genetic Screening of ZNF687 and PFN1 in a Paget's Disease of Bone Cohort Indicates an Important Role for the Nuclear Localization Signal of ZNF687Yentl Huybrechts, Raphaël De Ridder, Ellen Steenackers, et al.
The Journal of Clinical Endocrinology and Metabolism|January 4, 2024
A Mosaic Variant in CTNNB1/β-catenin as a Novel Cause for Osteopathia Striata With Cranial SclerosisYentl Huybrechts, Natasha M Appelman-Dijkstra, Ellen Steenackers, et al.
Calcified Tissue International|December 5, 2023
An Additional Lrp4 High Bone Mass Mutation Mitigates the Sost-Knockout Phenotype in Mice by Increasing Bone RemodelingGretl Hendrickx, Eveline Boudin, Ligia Mateiu, et al.
Pageof 3