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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 21, 2011
Mitochondrial reactive oxygen species are activated by mGluR5 through IP3 and activate ERK and PKA to increase excitability of amygdala neurons and pain behaviorZhen Li, Guangchen Ji, Volker Neugebauer
Molecular Pain|April 30, 2009
NR2B receptor blockade inhibits pain-related sensitization of amygdala neuronsGuangchen Ji, Csilla Horváth, Volker Neugebauer
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 22, 2016
Rescue of Impaired mGluR5-Driven Endocannabinoid Signaling Restores Prefrontal Cortical Output to Inhibit Pain in Arthritic RatsTakaki Kiritoshi, Guangchen Ji, Volker Neugebauer
Molecular Pain|February 16, 2013
Non-pain-related CRF1 activation in the amygdala facilitates synaptic transmission and pain responsesGuangchen Ji, Yu Fu, Hita Adwanikar, et al.
Molecular Pain|June 3, 2014
Nasal application of neuropeptide S inhibits arthritis pain-related behaviors through an action in the amygdalaGeorgina Medina, Guangchen Ji, Stéphanie Grégoire, et al.
Molecular Pain|June 7, 2007
Pain-related anxiety-like behavior requires CRF1 receptors in the amygdalaGuangchen Ji, Yu Fu, Katherine A Ruppert, et al.
International Journal of Molecular Sciences|August 12, 2023
Hmgb1 Silencing in the Amygdala Inhibits Pain-Related Behaviors in a Rat Model of Neuropathic PainPeyton Presto, Guangchen Ji, Olga Ponomareva, et al.
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