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Medrxiv : the Preprint Server for Health Sciences|February 20, 2025
Fusion oncoproteins and cooperating mutations define disease phenotypes in <i>NUP98</i>-rearranged leukemiaMasayuki Umeda, Ryan Hiltenbrand, Nicole L Michmerhuizen, et al.Blood|October 27, 2023
Acute myeloid leukemias with UBTF tandem duplications are sensitive to menin inhibitorsJuan M Barajas, Milad Rasouli, Masayuki Umeda, et al.Nature Communications|October 8, 2025
The genomic landscape of relapsed infant and childhood KMT2A-rearranged acute leukemiaLouise Ahlgren, Mattias Pilheden, Helena Sturesson, et al.Blood|July 23, 2025
Fusion Oncoproteins and Cooperating Mutations Define Disease Phenotypes in NUP98-Rearranged LeukemiaMasayuki Umeda, Ryan Lea Hiltenbrand, Nicole L Michmerhuizen, et al.Nature Communications|March 20, 2015
Rise and fall of subclones from diagnosis to relapse in pediatric B-acute lymphoblastic leukaemiaXiaotu Ma, Michael Edmonson, Donald Yergeau, et al.Blood Cancer Discovery|November 15, 2021
Integrative Genomic Analysis of Pediatric Myeloid-Related Acute Leukemias Identifies Novel Subtypes and Prognostic IndicatorsMaarten Fornerod, Jing Ma, Sanne Noort, et al.Nature Communications|April 5, 2023
Etiology of oncogenic fusions in 5,190 childhood cancers and its clinical and therapeutic implicationYanling Liu, Jonathon Klein, Richa Bajpai, et al.Nature Genetics|January 24, 2017
Pediatric non-Down syndrome acute megakaryoblastic leukemia is characterized by distinct genomic subsets with varying outcomesJasmijn D E de Rooij, Cristyn Branstetter, Jing Ma, et al.Nature Communications|April 9, 2014
The landscape of somatic mutations in epigenetic regulators across 1,000 paediatric cancer genomesRobert Huether, Li Dong, Xiang Chen, et al.Nature Genetics|April 16, 2013
Whole-genome sequencing identifies genetic alterations in pediatric low-grade gliomasJinghui Zhang, Gang Wu, Claudia P Miller, et al.Pageof 7