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International Journal of Molecular Sciences|December 29, 2019
Acadesine Circumvents Azacitidine Resistance in Myelodysplastic Syndrome and Acute Myeloid LeukemiaThomas Cluzeau, Nathan Furstoss, Coline Savy, et al.Nucleic Acids Research|February 14, 2019
Modulation of the ATM/autophagy pathway by a G-quadruplex ligand tips the balance between senescence and apoptosis in cancer cellsJennifer Beauvarlet, Paul Bensadoun, Elodie Darbo, et al.Oncotarget|May 18, 2017
BCL2L10 positive cells in bone marrow are an independent prognostic factor of azacitidine outcome in myelodysplastic syndrome and acute myeloid leukemiaValérie Vidal, Guillaume Robert, Laure Goursaud, et al.Experimental Hematology & Oncology|April 1, 2026
Dual targeting of GPX4 and TXNRD1 triggers eradication of AML cells through induction of apoptosis and ferroptosisCécile Favreau, Maxence Bourgoin, Coline Savy, et al.Autophagy|June 2, 2015
The PRKAA1/AMPKα1 pathway triggers autophagy during CSF1-induced human monocyte differentiation and is a potential target in CMMLSandrine Obba, Zoheir Hizir, Laurent Boyer, et al.The Journal of Investigative Dermatology|December 21, 2018
Caspase 1/11 Deficiency or Pharmacological Inhibition Mitigates Psoriasis-Like Phenotype in MiceLazaro Emilio Aira, Diogo Gonçalves, Jozef P Bossowski, et al.Cancer Research|June 8, 2021
Dual Covalent Inhibition of PKM and IMPDH Targets Metabolism in Cutaneous Metastatic MelanomaMarwa Zerhouni, Anthony R Martin, Nathan Furstoss, et al.Biorxiv : the Preprint Server for Biology|January 30, 2023
MCB-613 exploits a collateral sensitivity in drug resistant EGFR-mutant non-small cell lung cancer through covalent inhibition of KEAP1Christopher F Bassil, Gray R Anderson, Benjamin Mayro, et al.The Journal of Experimental Medicine|July 27, 2016
BCL-B (BCL2L10) is overexpressed in patients suffering from multiple myeloma (MM) and drives an MM-like disease in transgenic miceMohamed-Amine Hamouda, Arnaud Jacquel, Guillaume Robert, et al.Nature Communications|January 14, 2026
EGFR inhibitor-resistant lung cancers exhibit collateral sensitivity to a covalent, cysteine-independent KEAP1 oligomerizing molecular bridgeChristopher F Bassil, Kerry Dillon, Gray R Anderson, et al.Pageof 9