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Molecular Neurodegeneration|June 28, 2022
CX3CR1 deficiency aggravates amyloid driven neuronal pathology and cognitive decline in Alzheimer's diseaseShweta S Puntambekar, Miguel Moutinho, Peter Bor-Chian Lin, et al.Journal of Neurotrauma|September 29, 2015
Altered Neuroinflammation and Behavior after Traumatic Brain Injury in a Mouse Model of Alzheimer's DiseaseOlga Kokiko-Cochran, Lena Ransohoff, Mike Veenstra, et al.Molecular Neurodegeneration|October 18, 2017
TREM2 deficiency exacerbates tau pathology through dysregulated kinase signaling in a mouse model of tauopathyShane M Bemiller, Tyler J McCray, Kevin Allan, et al.Molecular Neurodegeneration|October 29, 2020
Trem2 Y38C mutation and loss of Trem2 impairs neuronal synapses in adult miceVaishnavi S Jadhav, Peter B C Lin, Taylor Pennington, et al.Journal of Neuroinflammation|September 27, 2018
Genetically enhancing the expression of chemokine domain of CX3CL1 fails to prevent tau pathology in mouse models of tauopathyShane M Bemiller, Nicole M Maphis, Shane V Formica, et al.Molecular Neurodegeneration|June 3, 2018
The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer's diseasePaul J Cheng-Hathaway, Erin G Reed-Geaghan, Taylor R Jay, et al.The Journal of Experimental Medicine|March 4, 2015
TREM2 deficiency eliminates TREM2+ inflammatory macrophages and ameliorates pathology in Alzheimer's disease mouse modelsTaylor R Jay, Crystal M Miller, Paul J Cheng, et al.Immunity|September 2, 2023
Genetic variants of phospholipase C-γ2 alter the phenotype and function of microglia and confer differential risk for Alzheimer's diseaseAndy P Tsai, Chuanpeng Dong, Peter Bor-Chian Lin, et al.Pageof 3