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European Thyroid Journal|September 24, 2024
IgG4 glycosylation contributes to the pathogenesis of IgG4 Hashimoto's thyroiditis via the complement pathwayChenxu Zhao, Zhiming Sun, Shuaihang Wang, et al.
Thyroid : Official Journal of the American Thyroid Association|September 3, 2013
A classification of Hashimoto's thyroiditis based on immunohistochemistry for IgG4 and IgGJing Zhang, Lanlan Zhao, Ying Gao, et al.
International Immunopharmacology|September 27, 2016
Increased proportions of Tc17 cells and NK cells may be risk factors for disease progression in Hashimoto's thyroiditisYalei Liu, Ran You, Nan Yu, et al.
International Immunopharmacology|November 28, 2017
CD26 expression is down-regulated on CD8+ T cells in patients with Hashimoto's thyroiditisYalei Liu, Yuan Li, Yan Gong, et al.
Molecular and Cellular Endocrinology|November 12, 2016
CD32b expression is down-regulated on double-negative memory B cells in patients with Hashimoto's thyroiditisYalei Liu, Yan Gong, Chenxue Qu, et al.
Cellular Immunology|February 12, 2015
The expression of Fcγ receptors in Hashimoto's thyroiditisYalei Liu, Mingming Liu, Yang Zhang, et al.
The Journal of Clinical Endocrinology and Metabolism|November 8, 2014
Changes in anti-thyroglobulin IgG glycosylation patterns in Hashimoto's thyroiditis patientsShanshan Yuan, Qianqian Li, Yang Zhang, et al.
European Thyroid Journal|May 13, 2021
Glycosylation of Anti-Thyroglobulin IgG1 and IgG4 Subclasses in Thyroid DiseasesYuan Li, Chenxu Zhao, Keli Zhao, et al.
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