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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 14, 2010
NADPH oxidase-dependent regulation of T-type Ca2+ channels and ryanodine receptors mediate the augmented exocytosis of catecholamines from intermittent hypoxia-treated neonatal rat chromaffin cellsDangjai Souvannakitti, Jayasri Nanduri, Guoxiang Yuan, et al.Journal of Cellular Physiology|February 9, 2011
Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxiaGuoxiang Yuan, Shakil A Khan, Weibo Luo, et al.Journal of Virology|February 14, 2020
Human Immunodeficiency Virus-Associated Exosomes Promote Kaposi's Sarcoma-Associated Herpesvirus Infection via the Epidermal Growth Factor ReceptorLechuang Chen, Zhimin Feng, Guoxiang Yuan, et al.The Journal of Physiology|April 27, 2004
Role of oxidative stress in intermittent hypoxia-induced immediate early gene activation in rat PC12 cellsGuoxiang Yuan, Gautam Adhikary, Andrew A McCormick, et al.American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|December 26, 2008
Reactive oxygen species-dependent endothelin signaling is required for augmented hypoxic sensory response of the neonatal carotid body by intermittent hypoxiaAnita Pawar, Jayasri Nanduri, Guoxiang Yuan, et al.American Journal of Physiology. Cell Physiology|July 29, 2010
Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cellsDangjai Souvannakitti, Barbara Kuri, Guoxiang Yuan, et al.Antioxidants & Redox Signaling|July 13, 2010
NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in ratsShakil A Khan, Jayasri Nanduri, Guoxiang Yuan, et al.American Journal of Physiology. Cell Physiology|November 8, 2014
CaV3.2 T-type Ca²⁺ channels in H₂S-mediated hypoxic response of the carotid bodyVladislav V Makarenko, Ying-Jie Peng, Guoxiang Yuan, et al.Toxins|August 25, 2023
Deciphering the Hazardous Effects of AFB1 and T-2 Toxins: Unveiling Toxicity and Oxidative Stress Mechanisms in PK15 Cells and Mouse KidneysShuai Xiao, Yingxin Wu, Suisui Gao, et al.American Journal of Physiology. Cell Physiology|January 31, 2014
Intermittent hypoxia-induced endothelial barrier dysfunction requires ROS-dependent MAP kinase activationVladislav V Makarenko, Peter V Usatyuk, Guoxiang Yuan, et al.Pageof 4