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Molecular Biology of the Cell|June 3, 2011
The Caenorhabditis elegans paxillin orthologue, PXL-1, is required for pharyngeal muscle contraction and for viabilityAdam Warner, Hiroshi Qadota, Guy M Benian, et al.Methods (San Diego, Calif.)|December 1, 2011
Bending amplitude - a new quantitative assay of C. elegans locomotion: identification of phenotypes for mutants in genes encoding muscle focal adhesion componentsJohn F Nahabedian, Hiroshi Qadota, Jeffrey N Stirman, et al.Molecular Biology of the Cell|September 1, 2007
Two LIM domain proteins and UNC-96 link UNC-97/pinch to myosin thick filaments in Caenorhabditis elegans muscleHiroshi Qadota, Kristina B Mercer, Rachel K Miller, et al.Scientific Reports|January 30, 2016
Muscle contraction phenotypic analysis enabled by optogenetics reveals functional relationships of sarcomere components in Caenorhabditis elegansHyundoo Hwang, Dawn E Barnes, Yohei Matsunaga, et al.Molecular Biology of the Cell|February 8, 2008
UNC-98 and UNC-96 interact with paramyosin to promote its incorporation into thick filaments of Caenorhabditis elegansRachel K Miller, Hiroshi Qadota, Kristina B Mercer, et al.Plos One|July 7, 2012
Large isoforms of UNC-89 (obscurin) are required for muscle cell architecture and optimal calcium release in Caenorhabditis elegansPatrick M Spooner, Jennifer Bonner, Andres V Maricq, et al.Molecular Biology of the Cell|June 14, 2023
FARL-11 (STRIP1/2) is required for sarcomere and sarcoplasmic reticulum organization in C. elegansSterling C T Martin, Hiroshi Qadota, Andres F Oberhauser, et al.Biorxiv : the Preprint Server for Biology|March 22, 2023
FARL-11 (STRIP1/2) is Required for Sarcomere and Sarcoplasmic Reticulum Organization in C. elegansSterling C T Martin, Hiroshi Qadota, Andres F Oberhauser, et al.The Journal of Biological Chemistry|April 3, 2014
Suppressor mutations suggest a surface on PAT-4 (Integrin-linked Kinase) that interacts with UNC-112 (Kindlin)Hiroshi Qadota, Yating Luo, Yohei Matsunaga, et al.Nature Communications|February 4, 2016
Diapause is associated with a change in the polarity of secretion of insulin-like peptidesYohei Matsunaga, Yoko Honda, Shuji Honda, et al.Pageof 8