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H Acha-Orbea

Showing results (71-80 of 97) with videos related to

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Journal of Virology|April 3, 1998
Immune response to mouse mammary tumor virus in mice lacking the alpha/beta interferon or the gamma interferon receptorI Maillard, P Launois, I Xenarios, et al.
Immunology|April 1, 1993
A comparative study of T-cell receptor V beta usage in non-obese diabetic (NOD) and I-E transgenic NOD miceN M Parish, H Acha-Orbea, E Simpson, et al.
Journal of Virology|October 6, 1997
Retrovirus-induced target cell activation in the early phases of infection: the mouse mammary tumor virus modelC Ardavin, F Luthi, M Andersson, et al.
European Journal of Immunology|March 1, 1995
T cell receptor V beta repertoire in mice lacking endogenous mouse mammary tumor provirusM Y Braun, E Jouvin-Marche, P N Marche, et al.
The Journal of Experimental Medicine|May 1, 1993
Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virusG A Waanders, A N Shakhov, W Held, et al.
Cell|August 13, 1993
Superantigen-induced immune stimulation amplifies mouse mammary tumor virus infection and allows virus transmissionW Held, G A Waanders, A N Shakhov, et al.
European Journal of Immunology|April 12, 2001
Functional plasticity of the LACK-reactive Vbeta4-Valpha8 CD4(+) T cells normally producing the early IL-4 instructing Th2 cell development and susceptibility to Leishmania major in BALB / c miceI Maillard, P Launois, H Himmelrich, et al.
Science (New York, N.Y.)|May 22, 1992
Less mortality but more relapses in experimental allergic encephalomyelitis in CD8-/- miceD R Koh, W P Fung-Leung, A Ho, et al.
The Journal of Experimental Medicine|February 1, 1993
Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virusW Held, A N Shakhov, S Izui, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 11, 1999
B cell response after MMTV infection: extrafollicular plasmablasts represent the main infected population and can transmit viral infectionC Ardavín, P Martín, I Ferrero, et al.
Pageof 10

Showing results (71-80 of 97) with videos related to

Sort By:
Pageof 10
Journal of Virology|April 3, 1998
Immune response to mouse mammary tumor virus in mice lacking the alpha/beta interferon or the gamma interferon receptorI Maillard, P Launois, I Xenarios, et al.
Immunology|April 1, 1993
A comparative study of T-cell receptor V beta usage in non-obese diabetic (NOD) and I-E transgenic NOD miceN M Parish, H Acha-Orbea, E Simpson, et al.
Journal of Virology|October 6, 1997
Retrovirus-induced target cell activation in the early phases of infection: the mouse mammary tumor virus modelC Ardavin, F Luthi, M Andersson, et al.
European Journal of Immunology|March 1, 1995
T cell receptor V beta repertoire in mice lacking endogenous mouse mammary tumor provirusM Y Braun, E Jouvin-Marche, P N Marche, et al.
The Journal of Experimental Medicine|May 1, 1993
Peripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virusG A Waanders, A N Shakhov, W Held, et al.
Cell|August 13, 1993
Superantigen-induced immune stimulation amplifies mouse mammary tumor virus infection and allows virus transmissionW Held, G A Waanders, A N Shakhov, et al.
European Journal of Immunology|April 12, 2001
Functional plasticity of the LACK-reactive Vbeta4-Valpha8 CD4(+) T cells normally producing the early IL-4 instructing Th2 cell development and susceptibility to Leishmania major in BALB / c miceI Maillard, P Launois, H Himmelrich, et al.
Science (New York, N.Y.)|May 22, 1992
Less mortality but more relapses in experimental allergic encephalomyelitis in CD8-/- miceD R Koh, W P Fung-Leung, A Ho, et al.
The Journal of Experimental Medicine|February 1, 1993
Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virusW Held, A N Shakhov, S Izui, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 11, 1999
B cell response after MMTV infection: extrafollicular plasmablasts represent the main infected population and can transmit viral infectionC Ardavín, P Martín, I Ferrero, et al.
Pageof 10