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Frontiers in Cellular Neuroscience|June 13, 2022
Neu1 Is Released From Activated Microglia, Stimulating Microglial Phagocytosis and Sensitizing Neurons to GlutamateDavid H Allendorf, Guy C BrownGlia|November 28, 2019
Activated microglia desialylate their surface, stimulating complement receptor 3-mediated phagocytosis of neuronsDavid H Allendorf, Mar Puigdellívol, Guy C BrownFrontiers in Cellular Neuroscience|June 26, 2020
Sialylation and Galectin-3 in Microglia-Mediated Neuroinflammation and NeurodegenerationMar Puigdellívol, David H Allendorf, Guy C BrownGlia|December 19, 2020
Sialylation acts as a checkpoint for innate immune responses in the central nervous systemChristine Klaus, Huan Liao, David H Allendorf, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 14, 2017
Activated Microglia Desialylate and Phagocytose Cells via Neuraminidase, Galectin-3, and Mer Tyrosine KinaseKoji Nomura, Anna Vilalta, David H Allendorf, et al.Journal of Neurochemistry|February 20, 2021
Microglial phagocytosis of neurons in neurodegeneration, and its regulationClaire A Butler, Alma S Popescu, Emily J A Kitchener, et al.Glia|December 8, 2022
Alzheimer's disease-associated R47H TREM2 increases, but wild-type TREM2 decreases, microglial phagocytosis of synaptosomes and neuronal lossAlma S Popescu, Claire A Butler, David H Allendorf, et al.Cell Reports|December 29, 2021
The microglial P2Y<sub>6</sub> receptor mediates neuronal loss and memory deficits in neurodegenerationMar Puigdellívol, Stefan Milde, Anna Vilalta, et al.The Journal of Biological Chemistry|April 6, 2021
Wild-type sTREM2 blocks Aβ aggregation and neurotoxicity, but the Alzheimer's R47H mutant increases Aβ aggregationAnna Vilalta, Ye Zhou, Jean Sevalle, et al.Pageof 1