Showing results (11-20 of 28) with videos related to
Sort By:
Pageof 3
Circulation Research|July 7, 2001
Molecular interactions between two long-QT syndrome gene products, HERG and KCNE2, rationalized by in vitro and in silico analysisR Mazhari, J L Greenstein, R L Winslow, et al.Advances in Experimental Medicine and Biology|January 1, 1995
Prospects for genetic manipulation of cardiac excitabilityJ H Lawrence, D C Johns, N Chiamvimonvat, et al.Biophysical Journal|January 5, 2000
Isoform-specific lidocaine block of sodium channels explained by differences in gatingH B Nuss, N G Kambouris, E Marbán, et al.The Journal of General Physiology|May 1, 1996
Functional consequences of lidocaine binding to slow-inactivated sodium channelsJ R Balser, H B Nuss, D N Romashko, et al.The Journal of General Physiology|December 1, 1995
Functional association of the beta 1 subunit with human cardiac (hH1) and rat skeletal muscle (mu 1) sodium channel alpha subunits expressed in Xenopus oocytesH B Nuss, N Chiamvimonvat, M T Pérez-García, et al.The American Journal of Physiology|January 1, 1991
Mechanical properties of adult feline ventricular myocytes in cultureP S Pollack, N L Carson, H B Nuss, et al.The Journal of Physiology|May 15, 2001
Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytesR A Li, J Miake, U C Hoppe, et al.The Journal of Clinical Investigation|August 1, 1995
Adenovirus-mediated expression of a voltage-gated potassium channel in vitro (rat cardiac myocytes) and in vivo (rat liver). A novel strategy for modifying excitabilityD C Johns, H B Nuss, N Chiamvimonvat, et al.The Journal of Physiology|July 15, 1996
External pore residue mediates slow inactivation in mu 1 rat skeletal muscle sodium channelsJ R Balser, H B Nuss, N Chiamvimonvat, et al.Circulation|March 12, 1998
Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channelN G Kambouris, H B Nuss, D C Johns, et al.Pageof 3