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The Journal of Physiology|July 15, 1996
Coupling between fast and slow inactivation revealed by analysis of a point mutation (F1304Q) in mu 1 rat skeletal muscle sodium channelsH B Nuss, J R Balser, D W Orias, et al.The Journal of Clinical Investigation|April 20, 2000
A revised view of cardiac sodium channel "blockade" in the long-QT syndromeN G Kambouris, H B Nuss, D C Johns, et al.Gene Therapy|October 1, 1996
Reversal of potassium channel deficiency in cells from failing hearts by adenoviral gene transfer: a prototype for gene therapy for disorders of cardiac excitability and contractilityH B Nuss, D C Johns, S Kääb, et al.Journal of the American College of Cardiology|August 1, 1997
Repolarization abnormalities, arrhythmia and sudden death in canine tachycardia-induced cardiomyopathyP H Pak, H B Nuss, R S Tunin, et al.Biophysical Journal|September 1, 1996
Single-channel analysis of inactivation-defective rat skeletal muscle sodium channels containing the F1304Q mutationJ H Lawrence, D W Orias, J R Balser, et al.Circulation Research|February 1, 1996
Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failureS Kääb, H B Nuss, N Chiamvimonvat, et al.The Journal of Clinical Investigation|December 15, 1996
Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channelsJ R Balser, H B Nuss, D W Orias, et al.Biophysical Journal|May 1, 1995
A mutation in the pore of the sodium channel alters gatingG F Tomaselli, N Chiamvimonvat, H B Nuss, et al.Pageof 3