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The Journal of Membrane Biology|November 1, 1992
Calcium-independent K(+)-selective channel from chromaffin granule membranesN Arispe, H B Pollard, E RojasProceedings of the National Academy of Sciences of the United States of America|January 15, 1993
Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminumN Arispe, E Rojas, H B PollardCellular and Molecular Neurobiology|October 1, 1995
Ion channel hypothesis for Alzheimer amyloid peptide neurotoxicityH B Pollard, N Arispe, E RojasProceedings of the National Academy of Sciences of the United States of America|October 1, 1996
Membrane fusion protein synexin (annexin VII) as a Ca2+/GTP sensor in exocytotic secretionH Caohuy, M Srivastava, H B PollardBiochimica Et Biophysica Acta|October 2, 1987
Effect of synexin on aggregation and fusion of chromaffin granule ghosts at pH 6S Nir, A Stutzin, H B PollardMolecular and Cellular Biochemistry|November 23, 1994
beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer diseaseN Arispe, H B Pollard, E RojasFEBS Letters|June 17, 1985
Real-time measurements of acetylcholine-induced release of ATP from bovine medullary chromaffin cellsE Rojas, H B Pollard, E HeldmanJournal of Neurochemistry|March 1, 1986
Stimulatory effect of ascorbic acid on norepinephrine biosynthesis in digitonin-permeabilized adrenal medullary chromaffin cellsK Morita, M Levine, H B PollardAnnals of the New York Academy of Sciences|September 24, 1993
A new hypothesis for the mechanism of amyloid toxicity, based on the calcium channel activity of amyloid beta protein (A beta P) in phospholipid bilayer membranesH B Pollard, E Rojas, N ArispeProceedings of the National Academy of Sciences of the United States of America|November 15, 1993
Giant multilevel cation channels formed by Alzheimer disease amyloid beta-protein [A beta P-(1-40)] in bilayer membranesN Arispe, H B Pollard, E RojasPageof 15