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Cancer Research|February 7, 2002
3-Methyladenine DNA glycosylase-deficient Aag null mice display unexpected bone marrow alkylation resistanceRichard B Roth, Leona D SamsonJournal of Bacteriology|November 5, 1999
Imbalanced base excision repair increases spontaneous mutation and alkylation sensitivity in Escherichia coliL M Posnick, L D SamsonProceedings of the National Academy of Sciences of the United States of America|September 15, 1999
Role of DNA mismatch repair and p53 in signaling induction of apoptosis by alkylating agentsM J Hickman, L D SamsonCarcinogenesis|April 28, 2000
Influence of DNA structure on hypoxanthine and 1,N(6)-ethenoadenine removal by murine 3-methyladenine DNA glycosylaseM D Wyatt, L D SamsonNeurology|August 1, 1977
Cerebral revascularization for transient ischemic attacksD Samson, C Watts, K ClarkMolecular Cell|April 8, 2004
Apoptotic signaling in response to a single type of DNA lesion, O(6)-methylguanineMark J Hickman, Leona D SamsonChemical Research in Toxicology|November 21, 2007
The protein degradation response of Saccharomyces cerevisiae to classical DNA-damaging agentsNicholas E Burgis, Leona D SamsonDNA Repair|January 8, 2013
Alkyladenine DNA glycosylase (AAG) localizes to mitochondria and interacts with mitochondrial single-stranded binding protein (mtSSB)Barbara van Loon, Leona D SamsonThe European Journal of Neuroscience|June 19, 2013
Impact of aging brain circuits on cognitionRachel D Samson, Carol A BarnesBlood|October 1, 1981
A simple in vitro method for the assessment of ineffective erythropoiesisD Samson, J Tikerpae, H CrownePageof 46