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Cell Cycle (Georgetown, Tex.)|April 8, 2010
Dominant roles of the Raf/MEK/ERK pathway in cell cycle progression, prevention of apoptosis and sensitivity to chemotherapeutic drugsLinda S Steelman, Stephen L Abrams, John G Shelton, et al.
Advances in Biological Regulation|November 4, 2015
Novel roles of androgen receptor, epidermal growth factor receptor, TP53, regulatory RNAs, NF-kappa-B, chromosomal translocations, neutrophil associated gelatinase, and matrix metalloproteinase-9 in prostate cancer and prostate cancer stem cellsWilliam H Chappell, Stephen L Abrams, Kvin Lertpiriyapong, et al.
Oncotarget|October 27, 2012
Effects of ectopic expression of NGAL on doxorubicin sensitivityWilliam H Chappell, Stephen L Abrams, Giuseppe Montalto, et al.
Virology|October 1, 1995
Identification and preliminary characterization of a chitinase gene in the Autographa californica nuclear polyhedrosis virus genomeR E Hawtin, K Arnold, M D Ayres, et al.
Current Pharmaceutical Design|October 22, 2013
Signaling intermediates (MAPK and PI3K) as therapeutic targets in NSCLCLudovica Ciuffreda, Ursula Cesta Incani, Linda S Steelman, et al.
Aging|June 4, 2020
Influences of TP53 and the anti-aging DDR1 receptor in controlling Raf/MEK/ERK and PI3K/Akt expression and chemotherapeutic drug sensitivity in prostate cancer cell linesWilliam H Chappell, Saverio Candido, Stephen L Abrams, et al.
Advances in Biological Regulation|October 6, 2020
Therapeutic resistance in breast cancer cells can result from deregulated EGFR signalingLinda S Steelman, William H Chappell, Shaw M Akula, et al.
Cell Cycle (Georgetown, Tex.)|May 4, 2010
The Raf/MEK/ERK pathway can govern drug resistance, apoptosis and sensitivity to targeted therapyStephen L Abrams, Linda S Steelman, John G Shelton, et al.
Oncotarget|July 7, 2011
Involvement of Akt-1 and mTOR in sensitivity of breast cancer to targeted therapyMelissa L Sokolosky, Kristin M Stadelman, William H Chappell, et al.
Cell Cycle (Georgetown, Tex.)|August 27, 2011
Involvement of Akt and mTOR in chemotherapeutic- and hormonal-based drug resistance and response to radiation in breast cancer cellsLinda S Steelman, Patrick Navolanic, William H Chappell, et al.
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