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Proceedings of the National Academy of Sciences of the United States of America|December 15, 1992
Scorpion toxins targeted against the sarcoplasmic reticulum Ca(2+)-release channel of skeletal and cardiac muscleH H Valdivia, M S Kirby, W J Lederer, et al.The Journal of Physiology|September 15, 1995
Modulation of cardiac ryanodine receptors of swine and rabbit by a phosphorylation-dephosphorylation mechanismA J Lokuta, T B Rogers, W J Lederer, et al.Science (New York, N.Y.)|March 31, 1995
Rapid adaptation of cardiac ryanodine receptors: modulation by Mg2+ and phosphorylationH H Valdivia, J H Kaplan, G C Ellis-Davies, et al.Biophysical Journal|November 1, 1994
Spatial non-uniformities in [Ca2+]i during excitation-contraction coupling in cardiac myocytesM B Cannell, H Cheng, W J LedererPflugers Archiv : European Journal of Physiology|October 1, 1994
Propagation of excitation-contraction coupling into ventricular myocytesH Cheng, M B Cannell, W J LedererScience (New York, N.Y.)|October 29, 1993
Calcium sparks: elementary events underlying excitation-contraction coupling in heart muscleH Cheng, W J Lederer, M B CannellScience (New York, N.Y.)|May 19, 1995
The control of calcium release in heart muscleM B Cannell, H Cheng, W J LedererCirculation Research|February 1, 1995
Partial inhibition of Ca2+ current by methoxyverapamil (D600) reveals spatial nonuniformities in [Ca2+]i during excitation-contraction coupling in cardiac myocytesH Cheng, M B Cannell, W J LedererScience (New York, N.Y.)|May 2, 1997
Defective excitation-contraction coupling in experimental cardiac hypertrophy and heart failureA M Gómez, H H Valdivia, H Cheng, et al.The Journal of Physiology|October 15, 1996
Ca2+ diffusion and sarcoplasmic reticulum transport both contribute to [Ca2+]i decline during Ca2+ sparks in rat ventricular myocytesA M Gómez, H Cheng, W J Lederer, et al.Pageof 331