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The Biochemical Journal|June 15, 1979
Unaltered catabolism of desialylated low-density lipoprotein in the pig and in cultured rat hepatocytesA D Attie, D B Weinstein, H H Freeze, et al.
Cancer Research|May 17, 2008
Inhibition of N-linked glycosylation disrupts receptor tyrosine kinase signaling in tumor cellsJoseph N Contessa, Mahaveer S Bhojani, Hudson H Freeze, et al.
Glycoconjugate Journal|September 26, 2000
Aglycone structure influences alpha-fucosyltransferase III activity using N-acetyllactosamine glycoside acceptorsY Miura, S Kim, J R Etchison, et al.
The Journal of Cell Biology|October 1, 1989
A Dictyostelium discoideum mutant that missorts and oversecretes lysosomal enzyme precursors is defective in endocytosisD L Ebert, H H Freeze, J Richardson, et al.
The Journal of Biological Chemistry|April 21, 1995
Identification of a novel glycosaminoglycan core-like molecule. I. 500 MHz 1H NMR analysis using a nano-NMR probe indicates the presence of a terminal alpha-GalNAc residue capping 4-methylumbelliferyl-beta-D-xylosidesA Manzi, P V Salimath, R C Spiro, et al.
The Journal of Clinical Endocrinology and Metabolism|April 21, 2005
Congenital disorder of glycosylation id presenting with hyperinsulinemic hypoglycemia and islet cell hyperplasiaLiangwu Sun, Erik A Eklund, Wendy K Chung, et al.
Acta Paediatrica (Oslo, Norway : 1992)|September 15, 1998
Failure of short-term mannose therapy of patients with carbohydrate-deficient glycoprotein syndrome type 1AS Kjaergaard, B Kristiansson, H Stibler, et al.
The Journal of Pediatrics|December 17, 2005
Clinical and biochemical characterization of a patient with congenital disorder of glycosylation (CDG) IIxYoshiaki Miura, Stacey K H Tay, Marion M Aw, et al.
Molecular Genetics and Metabolism|May 15, 2001
Genetic and metabolic analysis of the first adult with congenital disorder of glycosylation type Ib: long-term outcome and effects of mannose supplementationV Westphal, S Kjaergaard, J A Davis, et al.
The American Journal of Pathology|December 7, 2000
Reduced heparan sulfate accumulation in enterocytes contributes to protein-losing enteropathy in a congenital disorder of glycosylationV Westphal, S Murch, S Kim, et al.
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