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Biochemical Pharmacology
|
December 11, 1997
Sustained increase in intracellular free calcium and activation of cyclooxygenase-2 expression in mouse hepatoma cells treated with dioxin
A Puga, A Hoffer, S Zhou, et al.
Chemico-Biological Interactions
|
December 22, 1999
Inhibition of CYP1A1 enzyme activity in mouse hepatoma cell culture by soybean isoflavones
H G Shertzer, A Puga, C Chang, et al.
Biochemical Pharmacology
|
February 29, 2000
Activation of transcription factors activator protein-1 and nuclear factor-kappaB by 2,3,7,8-tetrachlorodibenzo-p-dioxin
A Puga, S J Barnes, C Chang, et al.
Experimental and Molecular Pathology
|
April 1, 1987
Protection against carbon tetrachloride hepatotoxicity by pretreatment with indole-3-carbinol
H G Shertzer, M P Niemi, F A Reitman, et al.
Biochemical and Biophysical Research Communications
|
December 14, 2001
Benzo[a]pyrene-induced toxicity: paradoxical protection in Cyp1a1(-/-) knockout mice having increased hepatic BaP-DNA adduct levels
S Uno, T P Dalton, H G Shertzer, et al.
American Journal of Physiology. Cell Physiology
|
March 14, 2001
Glutathione protects chemokine-scavenging and antioxidative defense functions in human RBCs
U J Dumaswala, L Zhuo, S Mahajan, et al.
Biochemical and Biophysical Research Communications
|
January 7, 2000
Targeted knockout of Cyp1a1 gene does not alter hepatic constitutive expression of other genes in the mouse [Ah] battery
T P Dalton, M Z Dieter, R S Matlib, et al.
Biochemical and Biophysical Research Communications
|
January 6, 1999
Dioxin causes a sustained oxidative stress response in the mouse
H G Shertzer, D W Nebert, A Puga, et al.
Carcinogenesis
|
October 1, 1994
Regulation of [Ah] gene battery enzymes and glutathione levels by 5,10-dihydroindeno[1,2-b]indole in mouse hepatoma cell lines
R M Liu, V Vasiliou, H Zhu, et al.
Cardiovascular Toxicology
|
September 6, 2002
Dioxin exposure is an environmental risk factor for ischemic heart disease
T P Dalton, J K Kerzee, B Wang, et al.
Page
of 6
Search research articles
Search
Showing results (51-60 of 60) with videos related to
Sort By:
Page
of 6
You have reached the last page of results.
This site can display upto 60 results.
Biochemical Pharmacology
|
December 11, 1997
Sustained increase in intracellular free calcium and activation of cyclooxygenase-2 expression in mouse hepatoma cells treated with dioxin
A Puga, A Hoffer, S Zhou, et al.
Chemico-Biological Interactions
|
December 22, 1999
Inhibition of CYP1A1 enzyme activity in mouse hepatoma cell culture by soybean isoflavones
H G Shertzer, A Puga, C Chang, et al.
Biochemical Pharmacology
|
February 29, 2000
Activation of transcription factors activator protein-1 and nuclear factor-kappaB by 2,3,7,8-tetrachlorodibenzo-p-dioxin
A Puga, S J Barnes, C Chang, et al.
Experimental and Molecular Pathology
|
April 1, 1987
Protection against carbon tetrachloride hepatotoxicity by pretreatment with indole-3-carbinol
H G Shertzer, M P Niemi, F A Reitman, et al.
Biochemical and Biophysical Research Communications
|
December 14, 2001
Benzo[a]pyrene-induced toxicity: paradoxical protection in Cyp1a1(-/-) knockout mice having increased hepatic BaP-DNA adduct levels
S Uno, T P Dalton, H G Shertzer, et al.
American Journal of Physiology. Cell Physiology
|
March 14, 2001
Glutathione protects chemokine-scavenging and antioxidative defense functions in human RBCs
U J Dumaswala, L Zhuo, S Mahajan, et al.
Biochemical and Biophysical Research Communications
|
January 7, 2000
Targeted knockout of Cyp1a1 gene does not alter hepatic constitutive expression of other genes in the mouse [Ah] battery
T P Dalton, M Z Dieter, R S Matlib, et al.
Biochemical and Biophysical Research Communications
|
January 6, 1999
Dioxin causes a sustained oxidative stress response in the mouse
H G Shertzer, D W Nebert, A Puga, et al.
Carcinogenesis
|
October 1, 1994
Regulation of [Ah] gene battery enzymes and glutathione levels by 5,10-dihydroindeno[1,2-b]indole in mouse hepatoma cell lines
R M Liu, V Vasiliou, H Zhu, et al.
Cardiovascular Toxicology
|
September 6, 2002
Dioxin exposure is an environmental risk factor for ischemic heart disease
T P Dalton, J K Kerzee, B Wang, et al.
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of 6