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Neuropeptides
|
July 1, 1991
The benzodiazepines: anxiolytic and withdrawal effects
H J Little
The Journal of Pharmacology and Experimental Therapeutics
|
September 1, 1995
Increases in non-N-methyl-D-aspartate glutamatergic transmission, but no change in gamma-aminobutyric acidB transmission, in CA1 neurons during withdrawal from in vivo chronic ethanol treatment
A Molleman, H J Little
Drug and Alcohol Dependence
|
July 28, 2004
Interactions between low concentrations of ethanol and nicotine on firing rate of ventral tegmental dopamine neurones
A Clark, H J Little
European Journal of Pharmacology
|
February 5, 1999
Increases in neuronal Ca2+ flux after withdrawal from chronic barbiturate treatment
M Rabbani, H J Little
Pharmacology, Biochemistry, and Behavior
|
May 20, 1998
CCK(B) antagonists protect against some aspects of the ethanol withdrawal syndrome
J Wilson, H J Little
Neuroscience
|
June 11, 1999
Effects of corticosterone on excitatory amino acid responses in dopamine-sensitive neurons in the ventral tegmental area
K Cho, H J Little
Brain Research
|
April 25, 2000
Differential effects of a dihydropyridine calcium channel antagonist on the components of ethanol tolerance
J W Smith, H J Little
Neuropharmacology
|
September 1, 1982
The benzodiazepine antagonist, Ro 15-1788, prevents the effects of flurazepam on the high pressure neurological syndrome
A R Bichard, H J Little
Neuropharmacology
|
April 30, 1999
Prolonged effects of chronic ethanol treatment on responses to repeated nicotine administration: interactions with environmental cues
W P Watson, H J Little
Psychopharmacology
|
January 1, 1995
Do GABAB receptors have a role in causing behavioural hyperexcitability, both during ethanol withdrawal and in naive mice?
A J Mead, H J Little
Page
of 12
Search research articles
Search
Showing results (11-20 of 113) with videos related to
Sort By:
Page
of 12
Neuropeptides
|
July 1, 1991
The benzodiazepines: anxiolytic and withdrawal effects
H J Little
The Journal of Pharmacology and Experimental Therapeutics
|
September 1, 1995
Increases in non-N-methyl-D-aspartate glutamatergic transmission, but no change in gamma-aminobutyric acidB transmission, in CA1 neurons during withdrawal from in vivo chronic ethanol treatment
A Molleman, H J Little
Drug and Alcohol Dependence
|
July 28, 2004
Interactions between low concentrations of ethanol and nicotine on firing rate of ventral tegmental dopamine neurones
A Clark, H J Little
European Journal of Pharmacology
|
February 5, 1999
Increases in neuronal Ca2+ flux after withdrawal from chronic barbiturate treatment
M Rabbani, H J Little
Pharmacology, Biochemistry, and Behavior
|
May 20, 1998
CCK(B) antagonists protect against some aspects of the ethanol withdrawal syndrome
J Wilson, H J Little
Neuroscience
|
June 11, 1999
Effects of corticosterone on excitatory amino acid responses in dopamine-sensitive neurons in the ventral tegmental area
K Cho, H J Little
Brain Research
|
April 25, 2000
Differential effects of a dihydropyridine calcium channel antagonist on the components of ethanol tolerance
J W Smith, H J Little
Neuropharmacology
|
September 1, 1982
The benzodiazepine antagonist, Ro 15-1788, prevents the effects of flurazepam on the high pressure neurological syndrome
A R Bichard, H J Little
Neuropharmacology
|
April 30, 1999
Prolonged effects of chronic ethanol treatment on responses to repeated nicotine administration: interactions with environmental cues
W P Watson, H J Little
Psychopharmacology
|
January 1, 1995
Do GABAB receptors have a role in causing behavioural hyperexcitability, both during ethanol withdrawal and in naive mice?
A J Mead, H J Little
Page
of 12