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The Journal of Biological Chemistry|April 11, 1997
Insulin-induced activation of NADPH-dependent H2O2 generation in human adipocyte plasma membranes is mediated by Galphai2H I Krieger-Brauer, P K Medda, H Kather
Metabolism: Clinical and Experimental|August 1, 1987
Mitigation of alimentary lipemia by postprandial exercise--phenomena and mechanismsG Schlierf, A Dinsenbacher, H Kather, et al.
European Journal of Clinical Investigation|February 1, 1985
Adrenergic regulation of lipolysis in abdominal adipocytes of obese subjects during caloric restriction: reversal of catecholamine action caused by relief of endogenous inhibitionH Kather, E Wieland, B Fischer, et al.
Journal of Bioluminescence and Chemiluminescence|July 1, 1989
Luminescence in the study of lipid metabolismE Wieland, D Niedmann, F Diedrich, et al.
The Journal of Biological Chemistry|January 25, 2000
Inhibitory effect of isoproterenol on NADPH-dependent H(2)O(2) generation in human adipocyte plasma membranes is mediated by betagamma-subunits derived from G(s)H I Krieger-Brauer, P K Medda, B Sattel, et al.
The Journal of Biological Chemistry|September 25, 1999
An antibody directed against residues 100-119 within the alpha-helical domain of Galpha(s) defines a novel contact site for beta-adrenergic receptorsH I Krieger-Brauer, P K Medda, U Hebling, et al.
The Journal of Clinical Investigation|October 1, 1985
Human fat cell lipolysis is primarily regulated by inhibitory modulators acting through distinct mechanismsH Kather, W Bieger, G Michel, et al.
Klinische Wochenschrift|February 1, 1988
Plasma-triglycerides and exercise: a delicate balanceG Schlierf, A Dinsenbacher, U Voggenreiter, et al.
The Journal of Clinical Investigation|August 1, 1987
Influences of variation in total energy intake and dietary composition on regulation of fat cell lipolysis in ideal-weight subjectsH Kather, E Wieland, A Scheurer, et al.
International Journal of Sports Medicine|August 1, 1988
Restricted alpha- and beta-adrenoceptor affinity of sulfoconjugated catecholamines in human mononuclear leukocytes, platelets, and fat cells and reduction of the postreceptor mechanismsE Werle, G Michel, T Lenz, et al.
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