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H Llewelyn Roderick

Showing results (81-90 of 103) with videos related to

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Life Science Alliance|September 20, 2024
PRDM16 determines specification of ventricular cardiomyocytes by suppressing alternative cell fatesJore Van Wauwe, Alexia Mahy, Sander Craps, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 27, 2009
The BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptorYi-Ping Rong, Geert Bultynck, Ademuyiwa S Aromolaran, et al.
The Journal of Cell Biology|July 13, 2005
CAPRI and RASAL impose different modes of information processing on Ras due to contrasting temporal filtering of Ca2+Qing Liu, Simon A Walker, Dingcheng Gao, et al.
Cardiovascular Research|April 19, 2018
Hyperactive ryanodine receptors in human heart failure and ischaemic cardiomyopathy reside outside of couplonsEef Dries, Demetrio J Santiago, Guillaume Gilbert, et al.
Briefings in Bioinformatics|July 24, 2025
Benchmarking of computational demultiplexing methods for single-nucleus RNA sequencing dataYile Fu, Mohamad Youness, Alessia Virzì, et al.
Journal of Molecular and Cellular Cardiology|February 23, 2018
Contractile responses to endothelin-1 are regulated by PKC phosphorylation of cardiac myosin binding protein-C in rat ventricular myocytesIoannis Smyrnias, Normann Goodwin, Dagmar Wachten, et al.
Journal of Molecular and Cellular Cardiology|April 1, 2023
InsP<sub>3</sub>R-RyR channel crosstalk augments sarcoplasmic reticulum Ca<sup>2+</sup> release and arrhythmogenic activity in post-MI pig cardiomyocytesXin Jin, Anna Meletiou, Joshua Chung, et al.
The Journal of Cell Biology|July 21, 2004
Bcl-2 functionally interacts with inositol 1,4,5-trisphosphate receptors to regulate calcium release from the ER in response to inositol 1,4,5-trisphosphateRui Chen, Ignacio Valencia, Fei Zhong, et al.
The Journal of Physiology|January 5, 2020
Altered adrenergic response in myocytes bordering a chronic myocardial infarction underlies in vivo triggered activity and repolarization instabilityEef Dries, Matthew Amoni, Bert Vandenberk, et al.
Journal of the American College of Cardiology|May 11, 2019
Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart FailureChandan K Nagaraju, Emma L Robinson, Mouna Abdesselem, et al.
Pageof 11

Showing results (81-90 of 103) with videos related to

Sort By:
Pageof 11
Life Science Alliance|September 20, 2024
PRDM16 determines specification of ventricular cardiomyocytes by suppressing alternative cell fatesJore Van Wauwe, Alexia Mahy, Sander Craps, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 27, 2009
The BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptorYi-Ping Rong, Geert Bultynck, Ademuyiwa S Aromolaran, et al.
The Journal of Cell Biology|July 13, 2005
CAPRI and RASAL impose different modes of information processing on Ras due to contrasting temporal filtering of Ca2+Qing Liu, Simon A Walker, Dingcheng Gao, et al.
Cardiovascular Research|April 19, 2018
Hyperactive ryanodine receptors in human heart failure and ischaemic cardiomyopathy reside outside of couplonsEef Dries, Demetrio J Santiago, Guillaume Gilbert, et al.
Briefings in Bioinformatics|July 24, 2025
Benchmarking of computational demultiplexing methods for single-nucleus RNA sequencing dataYile Fu, Mohamad Youness, Alessia Virzì, et al.
Journal of Molecular and Cellular Cardiology|February 23, 2018
Contractile responses to endothelin-1 are regulated by PKC phosphorylation of cardiac myosin binding protein-C in rat ventricular myocytesIoannis Smyrnias, Normann Goodwin, Dagmar Wachten, et al.
Journal of Molecular and Cellular Cardiology|April 1, 2023
InsP<sub>3</sub>R-RyR channel crosstalk augments sarcoplasmic reticulum Ca<sup>2+</sup> release and arrhythmogenic activity in post-MI pig cardiomyocytesXin Jin, Anna Meletiou, Joshua Chung, et al.
The Journal of Cell Biology|July 21, 2004
Bcl-2 functionally interacts with inositol 1,4,5-trisphosphate receptors to regulate calcium release from the ER in response to inositol 1,4,5-trisphosphateRui Chen, Ignacio Valencia, Fei Zhong, et al.
The Journal of Physiology|January 5, 2020
Altered adrenergic response in myocytes bordering a chronic myocardial infarction underlies in vivo triggered activity and repolarization instabilityEef Dries, Matthew Amoni, Bert Vandenberk, et al.
Journal of the American College of Cardiology|May 11, 2019
Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart FailureChandan K Nagaraju, Emma L Robinson, Mouna Abdesselem, et al.
Pageof 11