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Journal of Immunology (Baltimore, Md. : 1950)|August 8, 2008
Programmed death ligand 1 regulates a critical checkpoint for autoimmune myocarditis and pneumonitis in MRL miceJulie A Lucas, Julia Menke, Whitney A Rabacal, et al.
European Journal of Immunology|July 30, 2003
Recovery from EAE is associated with decreased survival of encephalitogenic T cells in the CNS of B7-1/B7-2-deficient miceTammy T Chang, Raymond A Sobel, Tao Wei, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 22, 2000
Intercellular adhesion molecule 1 is critical for activation of CD28-deficient T cellsJ L Gaglia, E A Greenfield, A Mattoo, et al.
Bio/Technology (Nature Publishing Company)|December 1, 1994
Detoxification of the plant toxin fluoroacetate by a genetically modified rumen bacteriumK Gregg, C L Cooper, D J Schafer, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 20, 2006
Blockade of CTLA-4 on CD4+CD25+ regulatory T cells abrogates their function in vivoSimon Read, Rebecca Greenwald, Ana Izcue, et al.
Journal of Immunology (Baltimore, Md. : 1950)|February 11, 1999
The role of CD80, CD86, and CTLA4 in alloimmune responses and the induction of long-term allograft survivalT A Judge, Z Wu, X G Zheng, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|July 9, 2008
Intrafollicular location of marginal zone/CD1d(hi) B cells is associated with autoimmune pathology in a mouse model of lupusBiyan Duan, Haitao Niu, Zhiwei Xu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 17, 2009
Enhanced selection of FoxP3+ T-regulatory cells protects CTLA-4-deficient mice from CNS autoimmune diseaseJohan Verhagen, Leona Gabrysová, Sophie Minaee, et al.
Health & Place|August 10, 2020
Close proximity to alcohol outlets is associated with increased crime and hazardous drinking: Pooled nationally representative data from New ZealandM Hobbs, L Marek, J Wiki, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 17, 2008
IL-10 and PD-L1 operate through distinct pathways to suppress T-cell activity during persistent viral infectionDavid G Brooks, Sang-Jun Ha, Heidi Elsaesser, et al.
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