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Haematologica|August 16, 2000

Immune reconstitution after autologous hematopoietic stem cell transplantation in relation to underlying disease, type of high-dose therapy and infectious complicationsH Steingrimsdottir, A Gruber, M Björkholm, et al.

Journal of Molecular Biology|January 20, 1991

Molecular analysis of ultraviolet-induced mutations in a xeroderma pigmentosum cell lineG Dorado, H Steingrimsdottir, C F Arlett, et al.

Oral Oncology|August 6, 1998

Prevalence of human papillomavirus infection in premalignant and malignant lesions of the oral cavity in U.K. subjects: a novel method of detectionF Elamin, H Steingrimsdottir, S Wanakulasuriya, et al.

Molecular & General Genetics : MGG|March 1, 1992

Mutant sequences in the rpsL gene of Escherichia coli B/r: mechanistic implications for spontaneous and ultraviolet light mutagenesisA R Timms, H Steingrimsdottir, A R Lehmann, et al.

Nucleic Acids Research|March 25, 1992

Mutations which alter splicing in the human hypoxanthine-guanine phosphoribosyltransferase geneH Steingrimsdottir, G Rowley, G Dorado, et al.

Nature Genetics|June 1, 1994

Mutations in the xeroderma pigmentosum group D DNA repair/transcription gene in patients with trichothiodystrophyB C Broughton, H Steingrimsdottir, C A Weber, et al.

Antimicrobial Agents and Chemotherapy|December 22, 1999

Bioavailability of aciclovir after oral administration of aciclovir and its prodrug valaciclovir to patients with leukopenia after chemotherapyH Steingrimsdottir, A Gruber, C Palm, et al.

Oncogene|May 18, 1995

U.v.-hypermutability of xeroderma pigmentosum cells demonstrated with a DNA-based mutation systemH Steingrimsdottir, D Beare, A M Carr, et al.

Mutation Research|March 1, 1989

Mutagenic treatments result in inactivation of expression of a transfected bacterial gene integrated into a human cell lineA R Lehmann, C F Arlett, S A Harcourt, et al.

International Journal of Oncology|February 10, 1998

p53 alterations and HPV infections are common in oral SCC: p53 gene mutations correlate with the absence of HPV 16-E6 DNAJ Penhallow, H Steingrimsdottir, F Elamin, et al.

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Showing results (1-10 of 22) with videos related to

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Haematologica|August 16, 2000

Immune reconstitution after autologous hematopoietic stem cell transplantation in relation to underlying disease, type of high-dose therapy and infectious complicationsH Steingrimsdottir, A Gruber, M Björkholm, et al.

Journal of Molecular Biology|January 20, 1991

Molecular analysis of ultraviolet-induced mutations in a xeroderma pigmentosum cell lineG Dorado, H Steingrimsdottir, C F Arlett, et al.

Oral Oncology|August 6, 1998

Prevalence of human papillomavirus infection in premalignant and malignant lesions of the oral cavity in U.K. subjects: a novel method of detectionF Elamin, H Steingrimsdottir, S Wanakulasuriya, et al.

Molecular & General Genetics : MGG|March 1, 1992

Mutant sequences in the rpsL gene of Escherichia coli B/r: mechanistic implications for spontaneous and ultraviolet light mutagenesisA R Timms, H Steingrimsdottir, A R Lehmann, et al.

Nucleic Acids Research|March 25, 1992

Mutations which alter splicing in the human hypoxanthine-guanine phosphoribosyltransferase geneH Steingrimsdottir, G Rowley, G Dorado, et al.

Nature Genetics|June 1, 1994

Mutations in the xeroderma pigmentosum group D DNA repair/transcription gene in patients with trichothiodystrophyB C Broughton, H Steingrimsdottir, C A Weber, et al.

Antimicrobial Agents and Chemotherapy|December 22, 1999

Bioavailability of aciclovir after oral administration of aciclovir and its prodrug valaciclovir to patients with leukopenia after chemotherapyH Steingrimsdottir, A Gruber, C Palm, et al.

Oncogene|May 18, 1995

U.v.-hypermutability of xeroderma pigmentosum cells demonstrated with a DNA-based mutation systemH Steingrimsdottir, D Beare, A M Carr, et al.

Mutation Research|March 1, 1989

Mutagenic treatments result in inactivation of expression of a transfected bacterial gene integrated into a human cell lineA R Lehmann, C F Arlett, S A Harcourt, et al.

International Journal of Oncology|February 10, 1998

p53 alterations and HPV infections are common in oral SCC: p53 gene mutations correlate with the absence of HPV 16-E6 DNAJ Penhallow, H Steingrimsdottir, F Elamin, et al.

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