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Trends in Immunology|September 15, 2004
More sympathy for autoimmunity with neuropeptide Y?Sammy Bedoui, Sachiko Miyake, Rainer H Straub, et al.Rheumatology (Oxford, England)|February 18, 2005
Predictive and potentially predictive factors in early arthritis: a multidisciplinary approachP Härle, T Bongartz, J Schölmerich, et al.Mechanisms of Ageing and Development|May 25, 2020
Absence of α-calcitonin gene-related peptide modulates bone remodeling properties of murine osteoblasts and osteoclasts in an age-dependent wayTanja Niedermair, Stephan Schirner, Mar Guaza Lasheras, et al.Alimentary Pharmacology & Therapeutics|February 4, 2003
Patients with refractory Crohn's disease or ulcerative colitis respond to dehydroepiandrosterone: a pilot studyT Andus, F Klebl, G Rogler, et al.Arthritis Research & Therapy|June 25, 2015
Reactivity of rat bone marrow-derived macrophages to neurotransmitter stimulation in the context of collagen II-induced arthritisDominique Muschter, Claudia Göttl, Mandy Vogel, et al.Arthritis and Rheumatism|June 4, 2005
Tumor necrosis factor inhibits conversion of dehydroepiandrosterone sulfate (DHEAS) to DHEA in rheumatoid arthritis synovial cells: a prerequisite for local androgen deficiencyClaudia Weidler, Sona Struharova, Martin Schmidt, et al.Annals of the Rheumatic Diseases|July 4, 2006
Quantitative determination of steroid hormone receptor positive cells in the synovium of patients with rheumatoid arthritis and osteoarthritis: is there a link to inflammation?S Capellino, B Riepl, L Rauch, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|July 6, 2000
A bacteria-induced switch of sympathetic effector mechanisms augments local inhibition of TNF-alpha and IL-6 secretion in the spleenR H Straub, H J Linde, D N Männel, et al.Journal of Leukocyte Biology|April 19, 2000
Neurotransmitters of the sympathetic nerve terminal are powerful chemoattractants for monocytesR H Straub, M Mayer, M Kreutz, et al.The Journal of Rheumatology|April 27, 2000
Altered function of the hypothalamic stress axes in patients with moderately active systemic lupus erythematosus. II. Dissociation between androstenedione, cortisol, or dehydroepiandrosterone and interleukin 6 or tumor necrosis factorB Zietz, T Reber, M Oertel, et al.Pageof 42