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The Journal of Biological Chemistry|March 30, 2001
Calcineurin enhances MAPK phosphatase-1 expression and p38 MAPK inactivation in cardiac myocytesH W Lim, L New, J Han, et al.Proceedings of the National Academy of Sciences of the United States of America|February 3, 2000
Targeted inhibition of calcineurin prevents agonist-induced cardiomyocyte hypertrophyT Taigen, L J De Windt, H W Lim, et al.The Journal of Biological Chemistry|May 2, 2000
Calcineurin promotes protein kinase C and c-Jun NH2-terminal kinase activation in the heart. Cross-talk between cardiac hypertrophic signaling pathwaysL J De Windt, H W Lim, S Haq, et al.Molecular and Cellular Biology|August 11, 2000
A calcineurin-NFATc3-dependent pathway regulates skeletal muscle differentiation and slow myosin heavy-chain expressionU Delling, J Tureckova, H W Lim, et al.Circulation Research|October 31, 2000
Calcineurin and beyond: cardiac hypertrophic signalingJ D MolkentinCirculation|May 24, 2000
Calcineurin expression, activation, and function in cardiac pressure-overload hypertrophyH W Lim, L J De Windt, L Steinberg, et al.Trends in Cardiovascular Medicine|January 11, 2001
Regulation of MEF2 by p38 MAPK and its implication in cardiomyocyte biologyJ Han, J D MolkentinJournal of Molecular and Cellular Cardiology|April 11, 2000
Reversal of cardiac hypertrophy in transgenic disease models by calcineurin inhibitionH W Lim, L J De Windt, J Mante, et al.Circulation Research|February 19, 2000
Calcineurin-mediated hypertrophy protects cardiomyocytes from apoptosis in vitro and in vivo: An apoptosis-independent model of dilated heart failureL J De Windt, H W Lim, T Taigen, et al.Circulation Research|January 5, 2001
The dual-specificity phosphatase MKP-1 limits the cardiac hypertrophic response in vitro and in vivoO F Bueno, L J De Windt, H W Lim, et al.Pageof 16