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Annual Review of Neuroscience|June 9, 2000
Glutamine repeats and neurodegenerationH Y Zoghbi, H T OrrCurrent Opinion in Neurobiology|October 6, 1999
Polyglutamine diseases: protein cleavage and aggregationH Y Zoghbi, H T OrrHuman Molecular Genetics|October 24, 2001
SCA1 molecular genetics: a history of a 13 year collaboration against glutaminesH T Orr, H Y ZoghbiMolecular Genetics and Metabolism|March 6, 1999
Pathogenesis of polyglutamine-induced disease: A model for SCA1I A Klement, H Y Zoghbi, H T OrrPhilosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 6, 1999
Progress in pathogenesis studies of spinocerebellar ataxia type 1C J Cummings, H T Orr, H Y ZoghbiNeurobiology of Disease|December 14, 2001
Reduction of Purkinje cell pathology in SCA1 transgenic mice by p53 deletionM D Shahbazian, H T Orr, H Y ZoghbiHuman Molecular Genetics|January 4, 2001
The spinocerebellar ataxia type 1 protein, ataxin-1, has RNA-binding activity that is inversely affected by the length of its polyglutamine tractS Yue, H G Serra, H Y Zoghbi, et al.Nature Neuroscience|January 29, 2000
Polyglutamine expansion down-regulates specific neuronal genes before pathologic changes in SCA1X Lin, B Antalffy, D Kang, et al.Nature Genetics|May 1, 1995
Expression analysis of the ataxin-1 protein in tissues from normal and spinocerebellar ataxia type 1 individualsA Servadio, B Koshy, D Armstrong, et al.Pageof 20