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American Journal of Physiology. Lung Cellular and Molecular Physiology|June 8, 2014
Reduced platelet-derived growth factor receptor expression is a primary feature of human bronchopulmonary dysplasiaAntonia P Popova, J Kelley Bentley, Tracy X Cui, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|December 4, 2012
Macrophage/epithelial cell CCL2 contributes to rhinovirus-induced hyperresponsiveness and inflammation in a mouse model of allergic airways diseaseDina Schneider, Jun Young Hong, Emily R Bowman, et al.
The Journal of Gene Medicine|March 28, 2001
Cationic liposome-mediated gene transfer to rat salivary epithelial cells in vitro and in vivoL Baccaglini, A T Shamsul Hoque, R B Wellner, et al.
Handbook of Experimental Pharmacology|December 20, 2008
Aquaporin-1 gene transfer to correct radiation-induced salivary hypofunctionBruce J Baum, Changyu Zheng, Ana P Cotrim, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|July 10, 2012
Glycogen synthase kinase-3β/β-catenin signaling regulates neonatal lung mesenchymal stromal cell myofibroblastic differentiationAntonia P Popova, J Kelley Bentley, Anuli C Anyanwu, et al.
Plos Pathogens|June 4, 2011
MDA5 and TLR3 initiate pro-inflammatory signaling pathways leading to rhinovirus-induced airways inflammation and hyperresponsivenessQiong Wang, David J Miller, Emily R Bowman, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|September 15, 2009
Elastase- and LPS-exposed mice display altered responses to rhinovirus infectionUmadevi Sajjan, Shyamala Ganesan, Adam T Comstock, et al.
Annals of the American Thoracic Society|March 20, 2014
Differential responses to rhinovirus- and influenza-associated pulmonary exacerbations in patients with cystic fibrosisIxsy A Ramirez, Lindsay J Caverly, Lindsay L Caverly, et al.
Gene Therapy|July 18, 2014
AdLTR2EF1α-FGF2-mediated prevention of fractionated irradiation-induced salivary hypofunction in swineL Guo, R Gao, J Xu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 24, 2004
Reengineered salivary glands are stable endogenous bioreactors for systemic gene therapeuticsAntonis Voutetakis, Marc R Kok, Changyu Zheng, et al.
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