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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 13, 2003
Signaling of hypoxia-induced autonomous proliferation of endothelial cellsM Schäfer, N Ewald, C Schäfer, et al.Pflugers Archiv : European Journal of Physiology|May 1, 1984
Absence of reoxygenation damage in isolated heart cells after anoxic injuryH M Piper, P Schwartz, R Spahr, et al.Basic Research in Cardiology|January 1, 1985
Development of new intercellular contacts between adult cardiac myocytes in cultureP Schwartz, H M Piper, R Spahr, et al.Cardiovascular Research|August 18, 2000
Mechanism of Ca(2+) overload in endothelial cells exposed to simulated ischemiaY Ladilov, C Schäfer, A Held, et al.Journal of Molecular and Cellular Cardiology|April 1, 1984
Early enzyme release from myocardial cells is not due to irreversible cell damageH M Piper, P Schwartz, R Spahr, et al.Advances in Experimental Medicine and Biology|January 1, 1992
NPY-(18-36) and pertussis toxin distinguish between the negative and positive contractile effects of neuropeptide Y (NPY) in ventricular cardiomyocytesB C Millar, B J McDermott, H M Piper, et al.Experimental Eye Research|August 1, 1990
Changes in the energy metabolism of cultured lens epithelial cells in comparison with the fresh lensH M Piper, R Spahr, A Krützfeldt, et al.Cardiovascular Research|October 28, 1999
Pretreatment with PKC activator protects cardiomyocytes against reoxygenation-induced hypercontracture independently of Ca2+ overloadY V Ladilov, C Balser-Schäfer, S Haffner, et al.Circulation Research|May 16, 2000
Expression, release, and biological activity of parathyroid hormone-related peptide from coronary endothelial cellsK Schlüter, C Katzer, K Frischkopf, et al.Biochimica Et Biophysica Acta|July 13, 1983
Fatty acid-membrane interactions in isolated cardiac mitochondria and erythrocytesH M Piper, O Sezer, P Schwartz, et al.Pageof 47