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Molecular Pharmacology|January 28, 2017
Structural Insights into the Atomistic Mechanisms of Action of Small Molecule Inhibitors Targeting the KCa3.1 Channel PoreHai M Nguyen, Vikrant Singh, Brandon Pressly, et al.
Molecular Pharmacology|February 1, 2014
Nanomolar bifenthrin alters synchronous Ca2+ oscillations and cortical neuron development independent of sodium channel activityZhengyu Cao, Yanjun Cui, Hai M Nguyen, et al.
Cellular and Molecular Life Sciences : CMLS|August 6, 2013
Domain structure and function of matrix metalloprotease 23 (MMP23): role in potassium channel traffickingCharles A Galea, Hai M Nguyen, K George Chandy, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 24, 2012
Modulation of voltage-gated K+ channels by the sodium channel β1 subunitHai M Nguyen, Haruko Miyazaki, Naoto Hoshi, et al.
Glia|October 4, 2016
Differential Kv1.3, KCa3.1, and Kir2.1 expression in "classically" and "alternatively" activated microgliaHai M Nguyen, Eva M Grössinger, Makoto Horiuchi, et al.
The Journal of Biological Chemistry|December 10, 2002
Molecular basis of isoform-specific micro-conotoxin block of cardiac, skeletal muscle, and brain Na+ channelsRonald A Li, Irene L Ennis, Tian Xue, et al.
The Journal of Biological Chemistry|January 10, 2013
Intracellular trafficking of the KV1.3 potassium channel is regulated by the prodomain of a matrix metalloproteaseHai M Nguyen, Charles A Galea, Galina Schmunk, et al.
Annals of Clinical and Translational Neurology|April 26, 2019
Repurposing the KCa3.1 inhibitor senicapoc for Alzheimer's diseaseLee-Way Jin, Jacopo Di Lucente, Hai M Nguyen, et al.
Neuropharmacology|November 27, 2022
Riluzole and novel naphthalenyl substituted aminothiazole derivatives prevent acute neural excitotoxic injury in a rat model of temporal lobe epilepsyThomas Kyllo, Vikrant Singh, Heesung Shim, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|December 15, 2015
The potassium channel KCa3.1 constitutes a pharmacological target for neuroinflammation associated with ischemia/reperfusion strokeYi-Je Chen, Hai M Nguyen, Izumi Maezawa, et al.
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