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Nature Immunology|August 8, 2006
Inhibition of transcription factor NF-kappaB in the central nervous system ameliorates autoimmune encephalomyelitis in miceGeert van Loo, Rossana De Lorenzi, Hauke Schmidt, et al.Cell Death and Differentiation|April 17, 2019
RIPK1 and death receptor signaling drive biliary damage and early liver tumorigenesis in mice with chronic hepatobiliary injurySantosh Krishna-Subramanian, Stephan Singer, Marietta Armaka, et al.The Ocular Surface|April 26, 2025
Dessicating stress triggers and exacerbates experimental ocular Graft-versus-host-diseaseUta Gehlsen, Martina Maass, Daniela Stary, et al.The Journal of Clinical Investigation|February 3, 2004
Conditional disruption of IkappaB kinase 2 fails to prevent obesity-induced insulin resistanceMathias Röhl, Manolis Pasparakis, Stephanie Baudler, et al.Proceedings of the National Academy of Sciences of the United States of America|April 9, 2004
Differential dependence of CD4+CD25+ regulatory and natural killer-like T cells on signals leading to NF-kappaB activationMarc Schmidt-Supprian, Jane Tian, Ethan P Grant, et al.The Journal of Clinical Investigation|August 4, 2006
Pathogenic role for skin macrophages in a mouse model of keratinocyte-induced psoriasis-like skin inflammationAthanasios Stratis, Manolis Pasparakis, Rudolf A Rupec, et al.Journal of Immunology (Baltimore, Md. : 1950)|November 12, 2010
Oligodendrocyte-specific FADD deletion protects mice from autoimmune-mediated demyelinationConor Mc Guire, Thomas Volckaert, Uta Wolke, et al.Human Molecular Genetics|January 10, 2006
Skin lesion development in a mouse model of incontinentia pigmenti is triggered by NEMO deficiency in epidermal keratinocytes and requires TNF signalingArianna Nenci, Marion Huth, Alfred Funteh, et al.Elife|December 3, 2014
Sharpin prevents skin inflammation by inhibiting TNFR1-induced keratinocyte apoptosisSnehlata Kumari, Younes Redouane, Jaime Lopez-Mosqueda, et al.Journal of Immunology (Baltimore, Md. : 1950)|July 22, 2004
I kappa B kinase 2 deficiency in T cells leads to defects in priming, B cell help, germinal center reactions, and homeostatic expansionMarc Schmidt-Supprian, Jane Tian, Hongbin Ji, et al.Pageof 24